{"type": "root", "children": [{"type": "p", "children": [{"type": "t", "text": "\n Although the provided abstract"}, {"type": "fg", "children": [{"type": "fg_f", "ref": "1"}]}, {"type": "t", "text": "does not explicitly mention or characterize CARS‐AS1, its discussion of category‐specific semantic impairment allows some inferences about how individual differences in premorbid expertise shape cognitive representations. In the study, patients with global semantic degradation nevertheless demonstrated selective preservation of domain‐specific knowledge—an automotive worker retained detailed car knowledge while a botanist retained plant knowledge—suggesting that early‐acquired expert representations are differentiated to such an extent that they resist overall degradation. In this context, if CARS‐AS1 were to be functionally related, one might hypothesize that it could play a modulatory role in preserving or influencing the neural substrates underlying expert concept representations. However, based solely on the data presented in this abstract, the specific functional implications or mechanisms attributed to CARS‐AS1 remain undetermined. Further research explicitly focusing on CARS‐AS1 would be required to clarify its role in modulating semantic networks and expertise‐related neural plasticity.\n "}]}, {"type": "rg", "children": [{"type": "r", "ref": 1, "children": [{"type": "t", "text": "Elizabeth Jefferies, Timothy T Rogers, Matthew A Lambon Ralph "}, {"type": "b", "children": [{"type": "t", "text": "Premorbid expertise produces category-specific impairment in a domain-general semantic disorder."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Neuropsychologia (2011)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1016/j.neuropsychologia.2011.07.024"}], "href": "https://doi.org/10.1016/j.neuropsychologia.2011.07.024"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "21816166"}], "href": "https://pubmed.ncbi.nlm.nih.gov/21816166"}]}]}]}