{"type": "root", "children": [{"type": "p", "children": [{"type": "t", "text": "\nThe provided study (PMID"}, {"type": "fg", "children": [{"type": "fg_f", "ref": "1"}]}, {"type": "t", "text": "focused on the roles of γ‐tubulin isoforms in microtubule nucleation, revealing that although γ‐tubulin 2 is capable of nucleating microtubules and compensating for the deficiency of γ‐tubulin 1 in cultured cells, its very low expression in mouse blastocysts explains why it cannot substitute for γ‐tubulin 1 during early embryonic development. The work highlights that functional redundancy in terms of nucleation activity exists, but the physiological impact is determined by the differential expression levels of the isoforms.\n \nIt should be noted that the abstract does not contain any information regarding SNORA8. Thus, based on the provided abstract, no conclusions about the function of SNORA8 can be drawn.\n"}]}, {"type": "rg", "children": [{"type": "r", "ref": 1, "children": [{"type": "t", "text": "Stanislav Vinopal, Markéta Cernohorská, Vadym Sulimenko, et al. "}, {"type": "b", "children": [{"type": "t", "text": "γ-Tubulin 2 nucleates microtubules and is downregulated in mouse early embryogenesis."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "PLoS One (2012)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1371/journal.pone.0029919"}], "href": "https://doi.org/10.1371/journal.pone.0029919"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "22235350"}], "href": "https://pubmed.ncbi.nlm.nih.gov/22235350"}]}]}]}