BCL9L Gene

Name	B-cell CLL/lymphoma 9-like
Description	Enables beta-catenin binding activity. Involved in several processes, including negative regulation of transforming growth factor beta receptor signaling pathway; positive regulation of epithelial to mesenchymal transition; and positive regulation of transcription by RNA polymerase II. Located in fibrillar center and nucleoplasm. Part of beta-catenin-TCF complex. [provided by Alliance of Genome Resources, Mar 2025]
Summary	{"type": "root", "children": [{"type": "p", "children": [{"type": "t", "text": "\n BCL9L (also referred to as BCL9‐2) is a pivotal coactivator within the canonical Wnt/β–catenin signaling cascade that integrates developmental cues with oncogenic processes in a tissue‐ and context‐dependent manner. In several cancer types, including colorectal, breast, pancreatic, and ovarian carcinomas, BCL9L binds to β–catenin to enhance the transcription of key Wnt target genes that govern cell proliferation, epithelial–mesenchymal transition (EMT), and stemness. For example, in colorectal cancer BCL9L promotes metastasis by facilitating Wnt‐driven gene expression (e.g., regulation of BAMBI)"}, {"type": "fg", "children": [{"type": "fg_f", "ref": "1"}]}, {"type": "t", "text": ", and its activity is counteracted by microRNAs such as miR‐29b, miR‐22, and miR‐214 to suppress EMT and tumor cell growth."}, {"type": "fg", "children": [{"type": "fg_f", "ref": "2"}]}, {"type": "t", "text": " In breast cancer, elevated BCL9L expression is associated with a more aggressive phenotype and has been shown to regulate estrogen receptor transcription via interactions with Sp1 independent of β–catenin binding, thereby impacting therapeutic responses."}, {"type": "fg", "children": [{"type": "fg_fs", "start_ref": "4", "end_ref": "6"}]}, {"type": "t", "text": " Moreover, in African American colorectal cancers that lack APC mutations, loss‐of‐function alterations in BCL9L delineate a distinct subtype with unique clinical and genetic features."}, {"type": "fg", "children": [{"type": "fg_f", "ref": "7"}]}, {"type": "t", "text": " Beyond its classical nuclear role in transcription, BCL9L can function in the cytoplasm to influence nontranscriptional processes such as odontogenesis, wherein it interacts with proteins involved in vesicular trafficking and enamel formation."}, {"type": "fg", "children": [{"type": "fg_f", "ref": "8"}]}, {"type": "t", "text": " In addition, BCL9L loss has been implicated in promoting chromosomal instability and enhanced aneuploidy tolerance by decreasing apoptotic regulators"}, {"type": "fg", "children": [{"type": "fg_f", "ref": "9"}]}, {"type": "t", "text": ", while its expression and activity are modulated by extrinsic factors—for instance, nerve growth factor can downregulate BCL9L to impact β–catenin signaling in ovarian cancer"}, {"type": "fg", "children": [{"type": "fg_f", "ref": "10"}]}, {"type": "t", "text": "and tumor suppressors like WWOX can inhibit its transcriptional function through disruption of β–catenin interactions."}, {"type": "fg", "children": [{"type": "fg_f", "ref": "11"}]}, {"type": "t", "text": " Developmentally, BCL9L is under the transcriptional control of Pax6, linking it to tissue homeostasis and organogenesis such as in the lens."}, {"type": "fg", "children": [{"type": "fg_f", "ref": "12"}]}, {"type": "t", "text": " Collectively, studies indicate that through its modulation of β–catenin–dependent and –independent pathways, BCL9L plays multifaceted roles in tumor progression, cell differentiation, and the maintenance of stem cell properties across diverse biological systems."}, {"type": "fg", "children": [{"type": "fg_fs", "start_ref": "14", "end_ref": "20"}]}, {"type": "t", "text": "\n "}]}, {"type": "rg", "children": [{"type": "r", "ref": 1, "children": [{"type": "t", "text": "Johannes Fritzmann, Markus Morkel, Daniel Besser, et al. "}, {"type": "b", "children": [{"type": "t", "text": "A colorectal cancer expression profile that includes transforming growth factor beta inhibitor BAMBI predicts metastatic potential."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Gastroenterology (2009)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1053/j.gastro.2009.03.041"}], "href": "https://doi.org/10.1053/j.gastro.2009.03.041"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "19328798"}], "href": "https://pubmed.ncbi.nlm.nih.gov/19328798"}]}, {"type": "r", "ref": 2, "children": [{"type": "t", "text": "Maitreyi Subramanian, Srinivasa R Rao, Pooja Thacker, et al. "}, {"type": "b", "children": [{"type": "t", "text": "MiR-29b downregulates canonical Wnt signaling by suppressing coactivators of β-catenin in human colorectal cancer cells."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "J Cell Biochem (2014)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1002/jcb.24869"}], "href": "https://doi.org/10.1002/jcb.24869"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "24913975"}], "href": "https://pubmed.ncbi.nlm.nih.gov/24913975"}]}, {"type": "r", "ref": 3, "children": [{"type": "t", "text": "Ruifang Sun, Zhigang Liu, Lin Han, et al. "}, {"type": "b", "children": [{"type": "t", "text": "miR-22 and miR-214 targeting BCL9L inhibit proliferation, metastasis, and epithelial-mesenchymal transition by down-regulating Wnt signaling in colon cancer."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "FASEB J (2019)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1096/fj.201801798RR"}], "href": "https://doi.org/10.1096/fj.201801798RR"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "30698996"}], "href": "https://pubmed.ncbi.nlm.nih.gov/30698996"}]}, {"type": "r", "ref": 4, "children": [{"type": "t", "text": "Nathalie Zatula, Maria Wiese, Jens Bunzendahl, et al. "}, {"type": "b", "children": [{"type": "t", "text": "The BCL9-2 proto-oncogene governs estrogen receptor alpha expression in breast tumorigenesis."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Oncotarget (2014)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.18632/oncotarget.2252"}], "href": "https://doi.org/10.18632/oncotarget.2252"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "25149534"}], "href": "https://pubmed.ncbi.nlm.nih.gov/25149534"}]}, {"type": "r", "ref": 5, "children": [{"type": "t", "text": "Hiroyuki Toya, Tetsunari Oyama, Susumu Ohwada, et al. "}, {"type": "b", "children": [{"type": "t", "text": "Immunohistochemical expression of the beta-catenin-interacting protein B9L is associated with histological high nuclear grade and immunohistochemical ErbB2/HER-2 expression in breast cancers."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Cancer Sci (2007)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1111/j.1349-7006.2007.00430.x"}], "href": "https://doi.org/10.1111/j.1349-7006.2007.00430.x"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "17309600"}], "href": "https://pubmed.ncbi.nlm.nih.gov/17309600"}]}, {"type": "r", "ref": 6, "children": [{"type": "t", "text": "Vida Vafaizadeh, David Buechel, Natalia Rubinstein, et al. "}, {"type": "b", "children": [{"type": "t", "text": "The interactions of Bcl9/Bcl9L with β-catenin and Pygopus promote breast cancer growth, invasion, and metastasis."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Oncogene (2021)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1038/s41388-021-02016-9"}], "href": "https://doi.org/10.1038/s41388-021-02016-9"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "34545187"}], "href": "https://pubmed.ncbi.nlm.nih.gov/34545187"}]}, {"type": "r", "ref": 7, "children": [{"type": "t", "text": "Rosa M Xicola, Zarko Manojlovic, Gaius J Augustus, et al. "}, {"type": "b", "children": [{"type": "t", "text": "Lack of APC somatic mutation is associated with early-onset colorectal cancer in African Americans."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Carcinogenesis (2018)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1093/carcin/bgy122"}], "href": "https://doi.org/10.1093/carcin/bgy122"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "30239619"}], "href": "https://pubmed.ncbi.nlm.nih.gov/30239619"}]}, {"type": "r", "ref": 8, "children": [{"type": "t", "text": "Claudio Cantù, Pierfrancesco Pagella, Tania D Shajiei, et al. "}, {"type": "b", "children": [{"type": "t", "text": "A cytoplasmic role of Wnt/β-catenin transcriptional cofactors Bcl9, Bcl9l, and Pygopus in tooth enamel formation."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Sci Signal (2017)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1126/scisignal.aah4598"}], "href": "https://doi.org/10.1126/scisignal.aah4598"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "28174279"}], "href": "https://pubmed.ncbi.nlm.nih.gov/28174279"}]}, {"type": "r", "ref": 9, "children": [{"type": "t", "text": "Carlos López-García, Laurent Sansregret, Enric Domingo, et al. "}, {"type": "b", "children": [{"type": "t", "text": "BCL9L Dysfunction Impairs Caspase-2 Expression Permitting Aneuploidy Tolerance in Colorectal Cancer."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Cancer Cell (2017)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1016/j.ccell.2016.11.001"}], "href": "https://doi.org/10.1016/j.ccell.2016.11.001"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "28073006"}], "href": "https://pubmed.ncbi.nlm.nih.gov/28073006"}]}, {"type": "r", "ref": 10, "children": [{"type": "t", "text": "Bo Li, Shaoxi Cai, Yi Zhao, et al. "}, {"type": "b", "children": [{"type": "t", "text": "Nerve growth factor modulates the tumor cells migration in ovarian cancer through the WNT/β-catenin pathway."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Oncotarget (2016)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.18632/oncotarget.13186"}], "href": "https://doi.org/10.18632/oncotarget.13186"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "27835587"}], "href": "https://pubmed.ncbi.nlm.nih.gov/27835587"}]}, {"type": "r", "ref": 11, "children": [{"type": "t", "text": "Perla El-Hage, Ambre Petitalot, Anne-Hélène Monsoro-Burq, et al. "}, {"type": "b", "children": [{"type": "t", "text": "The Tumor-Suppressor WWOX and HDAC3 Inhibit the Transcriptional Activity of the β-Catenin Coactivator BCL9-2 in Breast Cancer Cells."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Mol Cancer Res (2015)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1158/1541-7786.MCR-14-0180"}], "href": "https://doi.org/10.1158/1541-7786.MCR-14-0180"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "25678599"}], "href": "https://pubmed.ncbi.nlm.nih.gov/25678599"}]}, {"type": "r", "ref": 12, "children": [{"type": "t", "text": "Jian Sun, Shira Rockowitz, Qing Xie, et al. "}, {"type": "b", "children": [{"type": "t", "text": "Identification of in vivo DNA-binding mechanisms of Pax6 and reconstruction of Pax6-dependent gene regulatory networks during forebrain and lens development."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Nucleic Acids Res (2015)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1093/nar/gkv589"}], "href": "https://doi.org/10.1093/nar/gkv589"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "26138486"}], "href": "https://pubmed.ncbi.nlm.nih.gov/26138486"}]}, {"type": "r", "ref": 13, "children": [{"type": "t", "text": "Claudio Cantù, Dario Zimmerli, George Hausmann, et al. "}, {"type": "b", "children": [{"type": "t", "text": "Pax6-dependent, but β-catenin-independent, function of Bcl9 proteins in mouse lens development."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Genes Dev (2014)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1101/gad.246140.114"}], "href": "https://doi.org/10.1101/gad.246140.114"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "25184676"}], "href": "https://pubmed.ncbi.nlm.nih.gov/25184676"}]}, {"type": "r", "ref": 14, "children": [{"type": "t", "text": "Ken Matsuura, Takafumi Jigami, Kenzui Taniue, et al. "}, {"type": "b", "children": [{"type": "t", "text": "Identification of a link between Wnt/β-catenin signalling and the cell fusion pathway."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Nat Commun (2011)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1038/ncomms1551"}], "href": "https://doi.org/10.1038/ncomms1551"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "22109522"}], "href": "https://pubmed.ncbi.nlm.nih.gov/22109522"}]}, {"type": "r", "ref": 15, "children": [{"type": "t", "text": "Andrew S Brack, Fabienne Murphy-Seiler, Jasmine Hanifi, et al. "}, {"type": "b", "children": [{"type": "t", "text": "BCL9 is an essential component of canonical Wnt signaling that mediates the differentiation of myogenic progenitors during muscle regeneration."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Dev Biol (2009)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1016/j.ydbio.2009.08.014"}], "href": "https://doi.org/10.1016/j.ydbio.2009.08.014"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "19699733"}], "href": "https://pubmed.ncbi.nlm.nih.gov/19699733"}]}, {"type": "r", "ref": 16, "children": [{"type": "t", "text": "Yuriko Katoh, Masaru Katoh "}, {"type": "b", "children": [{"type": "t", "text": "FGF signaling inhibitor, SPRY4, is evolutionarily conserved target of WNT signaling pathway in progenitor cells."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Int J Mol Med (2006)"}]}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "16465403"}], "href": "https://pubmed.ncbi.nlm.nih.gov/16465403"}]}, {"type": "r", "ref": 17, "children": [{"type": "t", "text": "Felix H Brembeck, Maria Wiese, Nathalie Zatula, et al. "}, {"type": "b", "children": [{"type": "t", "text": "BCL9-2 promotes early stages of intestinal tumor progression."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Gastroenterology (2011)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1053/j.gastro.2011.06.039"}], "href": "https://doi.org/10.1053/j.gastro.2011.06.039"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "21703997"}], "href": "https://pubmed.ncbi.nlm.nih.gov/21703997"}]}, {"type": "r", "ref": 18, "children": [{"type": "t", "text": "Andreas E Moor, Pascale Anderle, Claudio Cantù, et al. "}, {"type": "b", "children": [{"type": "t", "text": "BCL9/9L-β-catenin Signaling is Associated With Poor Outcome in Colorectal Cancer."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "EBioMedicine (2015)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1016/j.ebiom.2015.10.030"}], "href": "https://doi.org/10.1016/j.ebiom.2015.10.030"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "26844272"}], "href": "https://pubmed.ncbi.nlm.nih.gov/26844272"}]}, {"type": "r", "ref": 19, "children": [{"type": "t", "text": "Xiaoshuang Wang, Mei Feng, Tengfei Xiao, et al. "}, {"type": "b", "children": [{"type": "t", "text": "BCL9/BCL9L promotes tumorigenicity through immune-dependent and independent mechanisms in triple negative breast cancer."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Oncogene (2021)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1038/s41388-021-01756-y"}], "href": "https://doi.org/10.1038/s41388-021-01756-y"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "33767438"}], "href": "https://pubmed.ncbi.nlm.nih.gov/33767438"}]}, {"type": "r", "ref": 20, "children": [{"type": "t", "text": "Giuseppina Sannino, Nicole Armbruster, Mona Bodenhöfer, et al. "}, {"type": "b", "children": [{"type": "t", "text": "Role of BCL9L in transforming growth factor-β (TGF-β)-induced epithelial-to-mesenchymal-transition (EMT) and metastasis of pancreatic cancer."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Oncotarget (2016)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.18632/oncotarget.12455"}], "href": "https://doi.org/10.18632/oncotarget.12455"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "27713160"}], "href": "https://pubmed.ncbi.nlm.nih.gov/27713160"}]}]}]}
Synonyms	BCL9-2, DLNB11
Proteins	BCL9L_HUMAN
NCBI Gene ID	283149
API
Download Associations
Predicted Functions
Co-expressed Genes
Expression in Tissues and Cell Lines

Functional Associations

BCL9L has 6,063 functional associations with biological entities spanning 8 categories (molecular profile, organism, functional term, phrase or reference, chemical, disease, phenotype or trait, structural feature, cell line, cell type or tissue, gene, protein or microRNA) extracted from 107 datasets.

Click the + buttons to view associations for BCL9L from the datasets below.

If available, associations are ranked by standardized value

Harmonizome 3.0

BCL9L Gene

Functional Associations

Please acknowledge the Harmonizome in your publications by citing the following reference(s):

	Dataset	Summary
	Achilles Cell Line Gene Essentiality Profiles	cell lines with fitness changed by BCL9L gene knockdown relative to other cell lines from the Achilles Cell Line Gene Essentiality Profiles dataset.
	Allen Brain Atlas Adult Human Brain Tissue Gene Expression Profiles	tissues with high or low expression of BCL9L gene relative to other tissues from the Allen Brain Atlas Adult Human Brain Tissue Gene Expression Profiles dataset.
	Allen Brain Atlas Adult Mouse Brain Tissue Gene Expression Profiles	tissues with high or low expression of BCL9L gene relative to other tissues from the Allen Brain Atlas Adult Mouse Brain Tissue Gene Expression Profiles dataset.
	Allen Brain Atlas Aging Dementia and Traumatic Brain Injury Tissue Sample Gene Expression Profiles	tissue samples with high or low expression of BCL9L gene relative to other tissue samples from the Allen Brain Atlas Aging Dementia and Traumatic Brain Injury Tissue Sample Gene Expression Profiles dataset.
	Allen Brain Atlas Developing Human Brain Tissue Gene Expression Profiles by Microarray	tissue samples with high or low expression of BCL9L gene relative to other tissue samples from the Allen Brain Atlas Developing Human Brain Tissue Gene Expression Profiles by Microarray dataset.
	Allen Brain Atlas Developing Human Brain Tissue Gene Expression Profiles by RNA-seq	tissue samples with high or low expression of BCL9L gene relative to other tissue samples from the Allen Brain Atlas Developing Human Brain Tissue Gene Expression Profiles by RNA-seq dataset.
	Allen Brain Atlas Prenatal Human Brain Tissue Gene Expression Profiles	tissues with high or low expression of BCL9L gene relative to other tissues from the Allen Brain Atlas Prenatal Human Brain Tissue Gene Expression Profiles dataset.
	BioGPS Human Cell Type and Tissue Gene Expression Profiles	cell types and tissues with high or low expression of BCL9L gene relative to other cell types and tissues from the BioGPS Human Cell Type and Tissue Gene Expression Profiles dataset.
	BioGPS Mouse Cell Type and Tissue Gene Expression Profiles	cell types and tissues with high or low expression of BCL9L gene relative to other cell types and tissues from the BioGPS Mouse Cell Type and Tissue Gene Expression Profiles dataset.
	CCLE Cell Line Gene CNV Profiles	cell lines with high or low copy number of BCL9L gene relative to other cell lines from the CCLE Cell Line Gene CNV Profiles dataset.
	CCLE Cell Line Gene Expression Profiles	cell lines with high or low expression of BCL9L gene relative to other cell lines from the CCLE Cell Line Gene Expression Profiles dataset.
	CCLE Cell Line Proteomics	Cell lines associated with BCL9L protein from the CCLE Cell Line Proteomics dataset.
	ChEA Transcription Factor Binding Site Profiles	transcription factor binding site profiles with transcription factor binding evidence at the promoter of BCL9L gene from the CHEA Transcription Factor Binding Site Profiles dataset.
	ChEA Transcription Factor Targets	transcription factors binding the promoter of BCL9L gene in low- or high-throughput transcription factor functional studies from the CHEA Transcription Factor Targets dataset.
	ChEA Transcription Factor Targets 2022	transcription factors binding the promoter of BCL9L gene in low- or high-throughput transcription factor functional studies from the CHEA Transcription Factor Targets 2022 dataset.
	CM4AI KOLF21J CRISPRi Gene Perturbation Atlas	gene perturbations changing expression of BCL9L gene from the CM4AI KOLF21J CRISPRi Gene Perturbation Atlas dataset.
	CM4AI U2OS Cell Map Protein Localization Assemblies	assemblies containing BCL9L protein from integrated AP-MS and IF data from the CM4AI U2OS Cell Map Protein Localization Assemblies dataset.
	COMPARTMENTS Curated Protein Localization Evidence Scores	cellular components containing BCL9L protein from the COMPARTMENTS Curated Protein Localization Evidence Scores dataset.
	COMPARTMENTS Curated Protein Localization Evidence Scores 2025	cellular components containing BCL9L protein from the COMPARTMENTS Curated Protein Localization Evidence Scores 2025 dataset.
	COMPARTMENTS Experimental Protein Localization Evidence Scores 2025	cellular components containing BCL9L protein in low- or high-throughput protein localization assays from the COMPARTMENTS Experimental Protein Localization Evidence Scores 2025 dataset.
	COMPARTMENTS Text-mining Protein Localization Evidence Scores	cellular components co-occuring with BCL9L protein in abstracts of biomedical publications from the COMPARTMENTS Text-mining Protein Localization Evidence Scores dataset.
	COMPARTMENTS Text-mining Protein Localization Evidence Scores 2025	cellular components co-occuring with BCL9L protein in abstracts of biomedical publications from the COMPARTMENTS Text-mining Protein Localization Evidence Scores 2025 dataset.
	COSMIC Cell Line Gene CNV Profiles	cell lines with high or low copy number of BCL9L gene relative to other cell lines from the COSMIC Cell Line Gene CNV Profiles dataset.
	COSMIC Cell Line Gene Mutation Profiles	cell lines with BCL9L gene mutations from the COSMIC Cell Line Gene Mutation Profiles dataset.
	CTD Gene-Chemical Interactions	chemicals interacting with BCL9L gene/protein from the curated CTD Gene-Chemical Interactions dataset.
	CTD Gene-Disease Associations	diseases associated with BCL9L gene/protein from the curated CTD Gene-Disease Associations dataset.
	DeepCoverMOA Drug Mechanisms of Action	small molecule perturbations with high or low expression of BCL9L protein relative to other small molecule perturbations from the DeepCoverMOA Drug Mechanisms of Action dataset.
	DepMap CRISPR Gene Dependency	cell lines with fitness changed by BCL9L gene knockdown relative to other cell lines from the DepMap CRISPR Gene Dependency dataset.
	DISEASES Experimental Gene-Disease Association Evidence Scores 2025	diseases associated with BCL9L gene in GWAS datasets from the DISEASES Experimental Gene-Disease Assocation Evidence Scores 2025 dataset.
	DISEASES Text-mining Gene-Disease Association Evidence Scores	diseases co-occuring with BCL9L gene in abstracts of biomedical publications from the DISEASES Text-mining Gene-Disease Assocation Evidence Scores dataset.
	DISEASES Text-mining Gene-Disease Association Evidence Scores 2025	diseases co-occuring with BCL9L gene in abstracts of biomedical publications from the DISEASES Text-mining Gene-Disease Assocation Evidence Scores 2025 dataset.
	DisGeNET Gene-Disease Associations	diseases associated with BCL9L gene in GWAS and other genetic association datasets from the DisGeNET Gene-Disease Associations dataset.
	DisGeNET Gene-Phenotype Associations	phenotypes associated with BCL9L gene in GWAS and other genetic association datasets from the DisGeNET Gene-Phenoptype Associations dataset.
	ENCODE Histone Modification Site Profiles	histone modification site profiles with high histone modification abundance at BCL9L gene from the ENCODE Histone Modification Site Profiles dataset.
	ENCODE Transcription Factor Binding Site Profiles	transcription factor binding site profiles with transcription factor binding evidence at the promoter of BCL9L gene from the ENCODE Transcription Factor Binding Site Profiles dataset.
	ENCODE Transcription Factor Targets	transcription factors binding the promoter of BCL9L gene in ChIP-seq datasets from the ENCODE Transcription Factor Targets dataset.
	ESCAPE Omics Signatures of Genes and Proteins for Stem Cells	PubMedIDs of publications reporting gene signatures containing BCL9L from the ESCAPE Omics Signatures of Genes and Proteins for Stem Cells dataset.
	GeneRIF Biological Term Annotations	biological terms co-occuring with BCL9L gene in literature-supported statements describing functions of genes from the GeneRIF Biological Term Annotations dataset.
	GeneSigDB Published Gene Signatures	PubMedIDs of publications reporting gene signatures containing BCL9L from the GeneSigDB Published Gene Signatures dataset.
	GEO Signatures of Differentially Expressed Genes for Diseases	disease perturbations changing expression of BCL9L gene from the GEO Signatures of Differentially Expressed Genes for Diseases dataset.