MYT1L Gene

HGNC Family	Zinc fingers
Name	myelin transcription factor 1-like
Description	This gene encodes a member of the zinc finger superfamily of transcription factors whose expression, thus far, has been found only in neuronal tissues. The encoded protein belongs to a novel class of cystein-cystein-histidine-cystein zinc finger proteins that function in the developing mammalian central nervous system. Forced expression of this gene in combination with the basic helix-loop-helix transcription factor NeuroD1 and the transcription factors POU class 3 homeobox 2 and achaete-scute family basic helix-loop-helix transcription factor 1 can convert fetal and postnatal human fibroblasts into induced neuronal cells, which are able to generate action potentials. Mutations in this gene have been associated with an autosomal dominant form of cognitive disability and with autism spectrum disorder. Alternative splicing results in multiple variants. [provided by RefSeq, Jul 2017]
Summary	{"type": "root", "children": [{"type": "p", "children": [{"type": "t", "text": "\nMYT1L is a neural‐specific zinc finger transcription factor that is highly expressed in postmitotic neurons and plays a pivotal role in promoting neuronal differentiation and maintaining neuronal identity. Functional studies have demonstrated that MYT1L is capable of reprogramming human fibroblasts into functional neurons and is essential for establishing proper neurodevelopmental programs. Its regulation appears critical in early brain development, as evidenced by its involvement in the establishment of mature neuronal phenotypes and in the repression of non‐neuronal gene programs."}, {"type": "fg", "children": [{"type": "fg_fs", "start_ref": "1", "end_ref": "4"}]}, {"type": "t", "text": "\n"}]}, {"type": "t", "text": "\n\n"}, {"type": "p", "children": [{"type": "t", "text": "\nClinical and genetic investigations have linked variants in MYT1L—whether due to rare copy number changes or point mutations—to a broad spectrum of neuropsychiatric and neurodevelopmental disorders. Loss‐of‐function mutations and deletions affecting MYT1L have been associated with intellectual disability, autism spectrum disorders, syndromic obesity, and disturbances in neuroendocrine function, whereas partial duplications of MYT1L are more frequently observed in patients with schizophrenia and related psychoses. Moreover, in the context of glioblastoma, re‐expression of MYT1L has been shown to suppress tumorigenesis by restraining oncogenic proliferation pathways."}, {"type": "fg", "children": [{"type": "fg_fs", "start_ref": "5", "end_ref": "14"}]}, {"type": "t", "text": "\n"}]}, {"type": "t", "text": "\n\n"}, {"type": "p", "children": [{"type": "t", "text": "\nMechanistically, MYT1L directly modulates gene expression by binding to DNA through its zinc finger domains and by recruiting transcriptional co‐regulators such as the Sin3L/Rpd3L histone deacetylase complex. It governs a network of helix–loop–helix transcription factors by repressing inhibitors like ID1 and by fine-tuning pathways such as WNT/NOTCH signaling. Deficiency in MYT1L perturbs these regulatory circuits, leading to altered neuronal maturation, dysregulated ion channel expression (for example, upregulation of SCN5A resulting in neuronal hyperactivity), and broader defects in neural homeostasis—effects that in experimental systems can be rescued by targeted genetic or pharmacologic intervention."}, {"type": "fg", "children": [{"type": "fg_fs", "start_ref": "15", "end_ref": "19"}]}, {"type": "t", "text": "\n"}]}, {"type": "rg", "children": [{"type": "r", "ref": 1, "children": [{"type": "t", "text": "Terry Vrijenhoek, Jacobine E Buizer-Voskamp, Inge van der Stelt, et al. "}, {"type": "b", "children": [{"type": "t", "text": "Recurrent CNVs disrupt three candidate genes in schizophrenia patients."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Am J Hum Genet (2008)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1016/j.ajhg.2008.09.011"}], "href": "https://doi.org/10.1016/j.ajhg.2008.09.011"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "18940311"}], "href": "https://pubmed.ncbi.nlm.nih.gov/18940311"}]}, {"type": "r", "ref": 2, "children": [{"type": "t", "text": "Servi J C Stevens, Conny M A van Ravenswaaij-Arts, Jannie W H Janssen, et al. 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"}, {"type": "b", "children": [{"type": "t", "text": "Analysis of transcriptional activity by the Myt1 and Myt1l transcription factors."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "J Cell Biochem (2018)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1002/jcb.26636"}], "href": "https://doi.org/10.1002/jcb.26636"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "29291346"}], "href": "https://pubmed.ncbi.nlm.nih.gov/29291346"}]}, {"type": "r", "ref": 4, "children": [{"type": "t", "text": "Li Ma, Hsien-yu Wang "}, {"type": "b", "children": [{"type": "t", "text": "Suppression of cyclic GMP-dependent protein kinase is essential to the Wnt/cGMP/Ca2+ pathway."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "J Biol Chem (2006)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1074/jbc.M603603200"}], "href": "https://doi.org/10.1074/jbc.M603603200"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "16920709"}], "href": "https://pubmed.ncbi.nlm.nih.gov/16920709"}]}, {"type": "r", "ref": 5, "children": [{"type": "t", "text": "Jian Hu, Allen L Ho, Liang Yuan, et al. "}, {"type": "b", "children": [{"type": "t", "text": "From the Cover: Neutralization of terminal differentiation in gliomagenesis."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Proc Natl Acad Sci U S A (2013)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1073/pnas.1308610110"}], "href": "https://doi.org/10.1073/pnas.1308610110"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "23918370"}], "href": "https://pubmed.ncbi.nlm.nih.gov/23918370"}]}, {"type": "r", "ref": 6, "children": [{"type": "t", "text": "Patricia Blanchet, Martina Bebin, Shaam Bruet, et al. "}, {"type": "b", "children": [{"type": "t", "text": "MYT1L mutations cause intellectual disability and variable obesity by dysregulating gene expression and development of the neuroendocrine hypothalamus."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "PLoS Genet (2017)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1371/journal.pgen.1006957"}], "href": "https://doi.org/10.1371/journal.pgen.1006957"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "28859103"}], "href": "https://pubmed.ncbi.nlm.nih.gov/28859103"}]}, {"type": "r", "ref": 7, "children": [{"type": "t", "text": "Juliette Coursimault, Anne-Marie Guerrot, Michelle M Morrow, et al. 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"}, {"type": "b", "children": [{"type": "t", "text": "Germline mosaic transmission of a novel duplication of PXDN and MYT1L to two male half-siblings with autism."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Psychiatr Genet (2012)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1097/YPG.0b013e32834dc3f5"}], "href": "https://doi.org/10.1097/YPG.0b013e32834dc3f5"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "22157634"}], "href": "https://pubmed.ncbi.nlm.nih.gov/22157634"}]}, {"type": "r", "ref": 9, "children": [{"type": "t", "text": "W Li, X Wang, J Zhao, et al. "}, {"type": "b", "children": [{"type": "t", "text": "Association study of myelin transcription factor 1-like polymorphisms with schizophrenia in Han Chinese population."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Genes Brain Behav (2012)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1111/j.1601-183X.2011.00734.x"}], "href": "https://doi.org/10.1111/j.1601-183X.2011.00734.x"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "21923761"}], "href": "https://pubmed.ncbi.nlm.nih.gov/21923761"}]}, {"type": "r", "ref": 10, "children": [{"type": "t", "text": "Ti Wang, Zhen Zeng, Tao Li, et al. 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"}, {"type": "b", "children": [{"type": "t", "text": "A novel MYT1L mutation in a patient with severe early-onset obesity and intellectual disability."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Am J Med Genet A (2018)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1002/ajmg.a.40370"}], "href": "https://doi.org/10.1002/ajmg.a.40370"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "30055078"}], "href": "https://pubmed.ncbi.nlm.nih.gov/30055078"}]}, {"type": "r", "ref": 12, "children": [{"type": "t", "text": "Patricia Mansfield, John N Constantino, Dustin Baldridge "}, {"type": "b", "children": [{"type": "t", "text": "MYT1L: A systematic review of genetic variation encompassing schizophrenia and autism."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Am J Med Genet B Neuropsychiatr Genet (2020)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1002/ajmg.b.32781"}], "href": "https://doi.org/10.1002/ajmg.b.32781"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "32267091"}], "href": "https://pubmed.ncbi.nlm.nih.gov/32267091"}]}, {"type": "r", "ref": 13, "children": [{"type": "t", "text": "Tiffany A Melhuish, Izabela Kowalczyk, Arkadi Manukyan, et al. "}, {"type": "b", "children": [{"type": "t", "text": "Myt1 and Myt1l transcription factors limit proliferation in GBM cells by repressing YAP1 expression."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Biochim Biophys Acta Gene Regul Mech (2018)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1016/j.bbagrm.2018.10.005"}], "href": "https://doi.org/10.1016/j.bbagrm.2018.10.005"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "30312684"}], "href": "https://pubmed.ncbi.nlm.nih.gov/30312684"}]}, {"type": "r", "ref": 14, "children": [{"type": "t", "text": "Elisabetta Tabolacci, Maria Grazia Pomponi, Laura Remondini, et al. "}, {"type": "b", "children": [{"type": "t", "text": "Co-Occurrence of Fragile X Syndrome with a Second Genetic Condition: Three Independent Cases of Double Diagnosis."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Genes (Basel) (2021)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.3390/genes12121909"}], "href": "https://doi.org/10.3390/genes12121909"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "34946857"}], "href": "https://pubmed.ncbi.nlm.nih.gov/34946857"}]}, {"type": "r", "ref": 15, "children": [{"type": "t", "text": "Agnieszka Kepa, Lourdes Martinez Medina, Susanne Erk, et al. "}, {"type": "b", "children": [{"type": "t", "text": "Associations of the Intellectual Disability Gene MYT1L with Helix-Loop-Helix Gene Expression, Hippocampus Volume and Hippocampus Activation During Memory Retrieval."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Neuropsychopharmacology (2017)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1038/npp.2017.91"}], "href": "https://doi.org/10.1038/npp.2017.91"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "28470180"}], "href": "https://pubmed.ncbi.nlm.nih.gov/28470180"}]}, {"type": "r", "ref": 16, "children": [{"type": "t", "text": "Bettina Weigel, Jana F Tegethoff, Sarah D Grieder, et al. "}, {"type": "b", "children": [{"type": "t", "text": "MYT1L haploinsufficiency in human neurons and mice causes autism-associated phenotypes that can be reversed by genetic and pharmacologic intervention."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Mol Psychiatry (2023)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1038/s41380-023-01959-7"}], "href": "https://doi.org/10.1038/s41380-023-01959-7"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "36782060"}], "href": "https://pubmed.ncbi.nlm.nih.gov/36782060"}]}, {"type": "r", "ref": 17, "children": [{"type": "t", "text": "Ryan Dale Marcum, Ishwar Radhakrishnan "}, {"type": "b", "children": [{"type": "t", "text": "The neuronal transcription factor Myt1L interacts via a conserved motif with the PAH1 domain of Sin3 to recruit the Sin3L/Rpd3L histone deacetylase complex."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "FEBS Lett (2020)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1002/1873-3468.13811"}], "href": "https://doi.org/10.1002/1873-3468.13811"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "32391601"}], "href": "https://pubmed.ncbi.nlm.nih.gov/32391601"}]}, {"type": "r", "ref": 18, "children": [{"type": "t", "text": "Keito Yoshimura, Yuji Morita, Kenji Konomi, et al. "}, {"type": "b", "children": [{"type": "t", "text": "A web-based survey on various symptoms of computer vision syndrome and the genetic understanding based on a multi-trait genome-wide association study."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Sci Rep (2021)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1038/s41598-021-88827-y"}], "href": "https://doi.org/10.1038/s41598-021-88827-y"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "33941792"}], "href": "https://pubmed.ncbi.nlm.nih.gov/33941792"}]}, {"type": "r", "ref": 19, "children": [{"type": "t", "text": "Yangmei Zhang, Haixia Zhu, Xunlei Zhang, et al. "}, {"type": "b", "children": [{"type": "t", "text": "Clinical significance of MYT1L gene polymorphisms in Chinese patients with gastric cancer."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "PLoS One (2013)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1371/journal.pone.0071979"}], "href": "https://doi.org/10.1371/journal.pone.0071979"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "24015200"}], "href": "https://pubmed.ncbi.nlm.nih.gov/24015200"}]}]}]}
Synonyms	myT1-L, ZC2HC4B, ZC2H2C2, NZF1, MRD39
Proteins	MYT1L_HUMAN
NCBI Gene ID	23040
API
Download Associations
Predicted Functions
Co-expressed Genes
Expression in Tissues and Cell Lines

Functional Associations

MYT1L has 5,074 functional associations with biological entities spanning 9 categories (molecular profile, organism, chemical, disease, phenotype or trait, functional term, phrase or reference, structural feature, cell line, cell type or tissue, gene, protein or microRNA, sequence feature) extracted from 121 datasets.

Click the + buttons to view associations for MYT1L from the datasets below.

If available, associations are ranked by standardized value

Harmonizome 3.0

MYT1L Gene

Functional Associations

Please acknowledge the Harmonizome in your publications by citing the following reference(s):

	Dataset	Summary
	Allen Brain Atlas Adult Human Brain Tissue Gene Expression Profiles	tissues with high or low expression of MYT1L gene relative to other tissues from the Allen Brain Atlas Adult Human Brain Tissue Gene Expression Profiles dataset.
	Allen Brain Atlas Adult Mouse Brain Tissue Gene Expression Profiles	tissues with high or low expression of MYT1L gene relative to other tissues from the Allen Brain Atlas Adult Mouse Brain Tissue Gene Expression Profiles dataset.
	Allen Brain Atlas Aging Dementia and Traumatic Brain Injury Tissue Sample Gene Expression Profiles	tissue samples with high or low expression of MYT1L gene relative to other tissue samples from the Allen Brain Atlas Aging Dementia and Traumatic Brain Injury Tissue Sample Gene Expression Profiles dataset.
	Allen Brain Atlas Developing Human Brain Tissue Gene Expression Profiles by Microarray	tissue samples with high or low expression of MYT1L gene relative to other tissue samples from the Allen Brain Atlas Developing Human Brain Tissue Gene Expression Profiles by Microarray dataset.
	Allen Brain Atlas Developing Human Brain Tissue Gene Expression Profiles by RNA-seq	tissue samples with high or low expression of MYT1L gene relative to other tissue samples from the Allen Brain Atlas Developing Human Brain Tissue Gene Expression Profiles by RNA-seq dataset.
	Allen Brain Atlas Prenatal Human Brain Tissue Gene Expression Profiles	tissues with high or low expression of MYT1L gene relative to other tissues from the Allen Brain Atlas Prenatal Human Brain Tissue Gene Expression Profiles dataset.
	BioGPS Cell Line Gene Expression Profiles	cell lines with high or low expression of MYT1L gene relative to other cell lines from the BioGPS Cell Line Gene Expression Profiles dataset.
	BioGPS Human Cell Type and Tissue Gene Expression Profiles	cell types and tissues with high or low expression of MYT1L gene relative to other cell types and tissues from the BioGPS Human Cell Type and Tissue Gene Expression Profiles dataset.
	BioGPS Mouse Cell Type and Tissue Gene Expression Profiles	cell types and tissues with high or low expression of MYT1L gene relative to other cell types and tissues from the BioGPS Mouse Cell Type and Tissue Gene Expression Profiles dataset.
	Carcinogenome Chemical Perturbation Carcinogenicity Signatures	small molecule perturbations changing expression of MYT1L gene from the Carcinogenome Chemical Perturbation Carcinogenicity Signatures dataset.
	CCLE Cell Line Gene CNV Profiles	cell lines with high or low copy number of MYT1L gene relative to other cell lines from the CCLE Cell Line Gene CNV Profiles dataset.
	CCLE Cell Line Gene Expression Profiles	cell lines with high or low expression of MYT1L gene relative to other cell lines from the CCLE Cell Line Gene Expression Profiles dataset.
	CellMarker Gene-Cell Type Associations	cell types associated with MYT1L gene from the CellMarker Gene-Cell Type Associations dataset.
	ChEA Transcription Factor Binding Site Profiles	transcription factor binding site profiles with transcription factor binding evidence at the promoter of MYT1L gene from the CHEA Transcription Factor Binding Site Profiles dataset.
	ChEA Transcription Factor Targets	transcription factors binding the promoter of MYT1L gene in low- or high-throughput transcription factor functional studies from the CHEA Transcription Factor Targets dataset.
	ChEA Transcription Factor Targets 2022	transcription factors binding the promoter of MYT1L gene in low- or high-throughput transcription factor functional studies from the CHEA Transcription Factor Targets 2022 dataset.
	ClinVar Gene-Phenotype Associations 2025	phenotypes associated with MYT1L gene from the curated ClinVar Gene-Phenotype Associations 2025 dataset.
	CM4AI KOLF21J CRISPRi Gene Perturbation Atlas	gene perturbations changing expression of MYT1L gene from the CM4AI KOLF21J CRISPRi Gene Perturbation Atlas dataset.
	CMAP Signatures of Differentially Expressed Genes for Small Molecules	small molecule perturbations changing expression of MYT1L gene from the CMAP Signatures of Differentially Expressed Genes for Small Molecules dataset.
	COMPARTMENTS Curated Protein Localization Evidence Scores	cellular components containing MYT1L protein from the COMPARTMENTS Curated Protein Localization Evidence Scores dataset.
	COMPARTMENTS Curated Protein Localization Evidence Scores 2025	cellular components containing MYT1L protein from the COMPARTMENTS Curated Protein Localization Evidence Scores 2025 dataset.
	COMPARTMENTS Text-mining Protein Localization Evidence Scores	cellular components co-occuring with MYT1L protein in abstracts of biomedical publications from the COMPARTMENTS Text-mining Protein Localization Evidence Scores dataset.
	COMPARTMENTS Text-mining Protein Localization Evidence Scores 2025	cellular components co-occuring with MYT1L protein in abstracts of biomedical publications from the COMPARTMENTS Text-mining Protein Localization Evidence Scores 2025 dataset.
	COSMIC Cell Line Gene CNV Profiles	cell lines with high or low copy number of MYT1L gene relative to other cell lines from the COSMIC Cell Line Gene CNV Profiles dataset.
	COSMIC Cell Line Gene Mutation Profiles	cell lines with MYT1L gene mutations from the COSMIC Cell Line Gene Mutation Profiles dataset.
	CTD Gene-Chemical Interactions	chemicals interacting with MYT1L gene/protein from the curated CTD Gene-Chemical Interactions dataset.
	CTD Gene-Disease Associations	diseases associated with MYT1L gene/protein from the curated CTD Gene-Disease Associations dataset.
	dbGAP Gene-Trait Associations	traits associated with MYT1L gene in GWAS and other genetic association datasets from the dbGAP Gene-Trait Associations dataset.
	DeepCoverMOA Drug Mechanisms of Action	small molecule perturbations with high or low expression of MYT1L protein relative to other small molecule perturbations from the DeepCoverMOA Drug Mechanisms of Action dataset.
	DepMap CRISPR Gene Dependency	cell lines with fitness changed by MYT1L gene knockdown relative to other cell lines from the DepMap CRISPR Gene Dependency dataset.
	DGIdb Drug Targets 2026	interacting drugs for MYT1L protein from the DGIdb Drug Targets 2026 dataset.
	DISEASES Curated Gene-Disease Association Evidence Scores 2025	diseases involving MYT1L gene from the DISEASES Curated Gene-Disease Association Evidence Scores 2025 dataset.
	DISEASES Experimental Gene-Disease Association Evidence Scores	diseases associated with MYT1L gene in GWAS datasets from the DISEASES Experimental Gene-Disease Assocation Evidence Scores dataset.
	DISEASES Text-mining Gene-Disease Association Evidence Scores	diseases co-occuring with MYT1L gene in abstracts of biomedical publications from the DISEASES Text-mining Gene-Disease Assocation Evidence Scores dataset.
	DISEASES Text-mining Gene-Disease Association Evidence Scores 2025	diseases co-occuring with MYT1L gene in abstracts of biomedical publications from the DISEASES Text-mining Gene-Disease Assocation Evidence Scores 2025 dataset.
	DisGeNET Gene-Disease Associations	diseases associated with MYT1L gene in GWAS and other genetic association datasets from the DisGeNET Gene-Disease Associations dataset.
	DisGeNET Gene-Phenotype Associations	phenotypes associated with MYT1L gene in GWAS and other genetic association datasets from the DisGeNET Gene-Phenoptype Associations dataset.
	ENCODE Histone Modification Site Profiles	histone modification site profiles with high histone modification abundance at MYT1L gene from the ENCODE Histone Modification Site Profiles dataset.
	ENCODE Transcription Factor Binding Site Profiles	transcription factor binding site profiles with transcription factor binding evidence at the promoter of MYT1L gene from the ENCODE Transcription Factor Binding Site Profiles dataset.
	ENCODE Transcription Factor Targets	transcription factors binding the promoter of MYT1L gene in ChIP-seq datasets from the ENCODE Transcription Factor Targets dataset.