NBAS Gene

Name	neuroblastoma amplified sequence
Description	This gene encodes a protein with two leucine zipper domains, a ribosomal protein S14 signature domain and a Sec39 like domain. The protein is thought to be involved in Golgi-to-ER transport. Mutations in this gene are associated with short stature, optic nerve atrophy, and Pelger-Huet anomaly. [provided by RefSeq, Oct 2012]
Summary	{"type": "root", "children": [{"type": "p", "children": [{"type": "t", "text": "\nNBAS is a multifunctional protein that plays a critical role in intracellular trafficking by serving as an integral subunit of the syntaxin‐18 complex. In this capacity, NBAS (also known as the neuroblastoma‐amplified gene, NAG) interacts with peripheral components such as p31, ZW10, RINT‐1, and others to mediate retrograde transport from the Golgi apparatus to the endoplasmic reticulum, thereby ensuring proper vesicle fusion and protein glycosylation. Disruption of this vesicular transport process has been implicated not only in aberrant glycosylation and Golgi recycling defects but also in chromosomal rearrangements observed in certain cancers, including neuroblastoma and acute leukemias."}, {"type": "fg", "children": [{"type": "fg_fs", "start_ref": "1", "end_ref": "9"}]}, {"type": "t", "text": "\n"}]}, {"type": "t", "text": "\n\n"}, {"type": "p", "children": [{"type": "t", "text": "\nIn addition to its role in vesicular trafficking, NBAS contributes to the regulation of mRNA quality control through participation in the nonsense‐mediated decay (NMD) pathway. Acting in concert with core NMD factors such as UPF1, NBAS helps modulate the abundance of transcripts—including those involved in cellular stress responses—and is an essential element of a conserved negative feedback loop that maintains NMD homeostasis across species."}, {"type": "fg", "children": [{"type": "fg_f", "ref": "10"}, {"type": "fg_f", "ref": "9"}]}, {"type": "t", "text": "\n"}]}, {"type": "t", "text": "\n\n"}, {"type": "p", "children": [{"type": "t", "text": "\nClinically, loss‐of‐function mutations in NBAS have been linked to a wide spectrum of multisystem disorders. Biallelic NBAS pathogenic variants have been associated with recurrent episodes of acute liver failure—often fever‐triggered—as well as with syndromes characterized by short stature, optic nerve atrophy, Pelger–Huët anomaly, and skeletal dysplasia (including osteogenesis imperfecta). Furthermore, cases with NBAS mutations have revealed immunologic defects, such as impaired natural killer cell cytotoxicity leading to hemophagocytic lymphohistiocytosis, and have even been identified in the context of neuroblastoma gene rearrangements. These findings underscore the broad impact of NBAS dysfunction on hepatic, skeletal, ocular, and immune systems, and suggest that disruption of its retrograde transport and mRNA surveillance activities underlies these diverse clinical phenotypes."}, {"type": "fg", "children": [{"type": "fg_fs", "start_ref": "11", "end_ref": "21"}]}, {"type": "t", "text": "\n"}]}, {"type": "rg", "children": [{"type": "r", "ref": 1, "children": [{"type": "t", "text": "Takehiro Aoki, Sarah Ichimura, Ayano Itoh, et al. "}, {"type": "b", "children": [{"type": "t", "text": "Identification of the neuroblastoma-amplified gene product as a component of the syntaxin 18 complex implicated in Golgi-to-endoplasmic reticulum retrograde transport."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Mol Biol Cell (2009)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1091/mbc.e08-11-1104"}], "href": "https://doi.org/10.1091/mbc.e08-11-1104"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "19369418"}], "href": "https://pubmed.ncbi.nlm.nih.gov/19369418"}]}, {"type": "r", "ref": 2, "children": [{"type": "t", "text": "Kazuhiro Fujita, Masashi Sanada, Hiroshi Harada, et al. "}, {"type": "b", "children": [{"type": "t", "text": "Molecular cloning of t(2;7)(p24.3;p14.2), a novel chromosomal translocation in myelodysplastic syndrome-derived acute myeloid leukemia."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "J Hum Genet (2009)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1038/jhg.2009.40"}], "href": "https://doi.org/10.1038/jhg.2009.40"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "19407829"}], "href": "https://pubmed.ncbi.nlm.nih.gov/19407829"}]}, {"type": "r", "ref": 3, "children": [{"type": "t", "text": "Valentina Boeva, Stéphanie Jouannet, Romain Daveau, et al. "}, {"type": "b", "children": [{"type": "t", "text": "Breakpoint features of genomic rearrangements in neuroblastoma with unbalanced translocations and chromothripsis."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "PLoS One (2013)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1371/journal.pone.0072182"}], "href": "https://doi.org/10.1371/journal.pone.0072182"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "23991058"}], "href": "https://pubmed.ncbi.nlm.nih.gov/23991058"}]}, {"type": "r", "ref": 4, "children": [{"type": "t", "text": "Tobias B Haack, Christian Staufner, Marlies G Köpke, et al. "}, {"type": "b", "children": [{"type": "t", "text": "Biallelic Mutations in NBAS Cause Recurrent Acute Liver Failure with Onset in Infancy."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Am J Hum Genet (2015)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1016/j.ajhg.2015.05.009"}], "href": "https://doi.org/10.1016/j.ajhg.2015.05.009"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "26073778"}], "href": "https://pubmed.ncbi.nlm.nih.gov/26073778"}]}, {"type": "r", "ref": 5, "children": [{"type": "t", "text": "Nuria Garcia Segarra, Diana Ballhausen, Heather Crawford, et al. "}, {"type": "b", "children": [{"type": "t", "text": "NBAS mutations cause a multisystem disorder involving bone, connective tissue, liver, immune system, and retina."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Am J Med Genet A (2015)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1002/ajmg.a.37338"}], "href": "https://doi.org/10.1002/ajmg.a.37338"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "26286438"}], "href": "https://pubmed.ncbi.nlm.nih.gov/26286438"}]}, {"type": "r", "ref": 6, "children": [{"type": "t", "text": "Eleonora Palagano, Giulia Zuccarini, Paolo Prontera, et al. "}, {"type": "b", "children": [{"type": "t", "text": "Mutations in the Neuroblastoma Amplified Sequence gene in a family affected by Acrofrontofacionasal Dysostosis type 1."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Bone (2018)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1016/j.bone.2018.06.013"}], "href": "https://doi.org/10.1016/j.bone.2018.06.013"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "29929043"}], "href": "https://pubmed.ncbi.nlm.nih.gov/29929043"}]}, {"type": "r", "ref": 7, "children": [{"type": "t", "text": "Diana Carli, Elisa Giorgio, Francesca Pantaleoni, et al. "}, {"type": "b", "children": [{"type": "t", "text": "NBAS pathogenic variants: Defining the associated clinical and facial phenotype and genotype-phenotype correlations."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Hum Mutat (2019)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1002/humu.23734"}], "href": "https://doi.org/10.1002/humu.23734"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "30825388"}], "href": "https://pubmed.ncbi.nlm.nih.gov/30825388"}]}, {"type": "r", "ref": 8, "children": [{"type": "t", "text": "Francisco Javier Cotrina-Vinagre, María Elena Rodríguez-García, Elena Martín-Hernández, et al. "}, {"type": "b", "children": [{"type": "t", "text": "Characterization of a complex phenotype (fever-dependent recurrent acute liver failure and osteogenesis imperfecta) due to NBAS and P4HB variants."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Mol Genet Metab (2021)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1016/j.ymgme.2021.02.007"}], "href": "https://doi.org/10.1016/j.ymgme.2021.02.007"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "33707149"}], "href": "https://pubmed.ncbi.nlm.nih.gov/33707149"}]}, {"type": "r", "ref": 9, "children": [{"type": "t", "text": "Valentina Granata, Isabel Pagani, Emanuela Morenghi, et al. "}, {"type": "b", "children": [{"type": "t", "text": "Modulation of NBAS-Related Functions in the Early Response to SARS-CoV-2 Infection."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Int J Mol Sci (2023)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.3390/ijms24032634"}], "href": "https://doi.org/10.3390/ijms24032634"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "36768954"}], "href": "https://pubmed.ncbi.nlm.nih.gov/36768954"}]}, {"type": "r", "ref": 10, "children": [{"type": "t", "text": "Dasa Longman, Nele Hug, Marianne Keith, et al. "}, {"type": "b", "children": [{"type": "t", "text": "DHX34 and NBAS form part of an autoregulatory NMD circuit that regulates endogenous RNA targets in human cells, zebrafish and Caenorhabditis elegans."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Nucleic Acids Res (2013)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1093/nar/gkt585"}], "href": "https://doi.org/10.1093/nar/gkt585"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "23828042"}], "href": "https://pubmed.ncbi.nlm.nih.gov/23828042"}]}, {"type": "r", "ref": 11, "children": [{"type": "t", "text": "Setsuko Kaneko, Miki Ohira, Yohko Nakamura, et al. "}, {"type": "b", "children": [{"type": "t", "text": "Relationship of DDX1 and NAG gene amplification/overexpression to the prognosis of patients with MYCN-amplified neuroblastoma."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "J Cancer Res Clin Oncol (2007)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1007/s00432-006-0156-y"}], "href": "https://doi.org/10.1007/s00432-006-0156-y"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "17028906"}], "href": "https://pubmed.ncbi.nlm.nih.gov/17028906"}]}, {"type": "r", "ref": 12, "children": [{"type": "t", "text": "Nadezda Maksimova, Kenju Hara, Irina Nikolaeva, et al. "}, {"type": "b", "children": [{"type": "t", "text": "Neuroblastoma amplified sequence gene is associated with a novel short stature syndrome characterised by optic nerve atrophy and Pelger-Huët anomaly."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "J Med Genet (2010)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1136/jmg.2009.074815"}], "href": "https://doi.org/10.1136/jmg.2009.074815"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "20577004"}], "href": "https://pubmed.ncbi.nlm.nih.gov/20577004"}]}, {"type": "r", "ref": 13, "children": [{"type": "t", "text": "M Balasubramanian, J Hurst, S Brown, et al. "}, {"type": "b", "children": [{"type": "t", "text": "Compound heterozygous variants in NBAS as a cause of atypical osteogenesis imperfecta."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Bone (2017)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1016/j.bone.2016.10.023"}], "href": "https://doi.org/10.1016/j.bone.2016.10.023"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "27789416"}], "href": "https://pubmed.ncbi.nlm.nih.gov/27789416"}]}, {"type": "r", "ref": 14, "children": [{"type": "t", "text": "Frederico S Regateiro, Serkan Belkaya, Nélson Neves, et al. "}, {"type": "b", "children": [{"type": "t", "text": "Recurrent elevated liver transaminases and acute liver failure in two siblings with novel bi-allelic mutations of NBAS."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Eur J Med Genet (2017)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1016/j.ejmg.2017.05.005"}], "href": "https://doi.org/10.1016/j.ejmg.2017.05.005"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "28576691"}], "href": "https://pubmed.ncbi.nlm.nih.gov/28576691"}]}, {"type": "r", "ref": 15, "children": [{"type": "t", "text": "Jia-Qi Li, Yi-Ling Qiu, Jing-Yu Gong, et al. "}, {"type": "b", "children": [{"type": "t", "text": "Novel NBAS mutations and fever-related recurrent acute liver failure in Chinese children: a retrospective study."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "BMC Gastroenterol (2017)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1186/s12876-017-0636-3"}], "href": "https://doi.org/10.1186/s12876-017-0636-3"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "28629372"}], "href": "https://pubmed.ncbi.nlm.nih.gov/28629372"}]}, {"type": "r", "ref": 16, "children": [{"type": "t", "text": "N R Maksimova, A N Nogovicina, Kh A Kurtanov, et al. "}, {"type": "b", "children": [{"type": "t", "text": "[Population frequency and age of mutation G5741→A in gene NBAS which is a cause of SOPH syndrome in Sakha (Yakutia) Republic]."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Genetika (2016)"}]}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "29369590"}], "href": "https://pubmed.ncbi.nlm.nih.gov/29369590"}]}, {"type": "r", "ref": 17, "children": [{"type": "t", "text": "J Chavany, A Cano, B Roquelaure, et al. "}, {"type": "b", "children": [{"type": "t", "text": "Mutations in NBAS and SCYL1, genetic causes of recurrent liver failure in children: Three case reports and a literature review."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Arch Pediatr (2020)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1016/j.arcped.2020.01.003"}], "href": "https://doi.org/10.1016/j.arcped.2020.01.003"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "32146038"}], "href": "https://pubmed.ncbi.nlm.nih.gov/32146038"}]}, {"type": "r", "ref": 18, "children": [{"type": "t", "text": "Yves Lacassie, Britt Johnson, Guillermo Lay-Son, et al. 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"}, {"type": "b", "children": [{"type": "t", "text": "NBAS Variants Are Associated with Quantitative and Qualitative NK and B Cell Deficiency."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "J Clin Immunol (2021)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1007/s10875-021-01110-7"}], "href": "https://doi.org/10.1007/s10875-021-01110-7"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "34386911"}], "href": "https://pubmed.ncbi.nlm.nih.gov/34386911"}]}, {"type": "r", "ref": 21, "children": [{"type": "t", "text": "Xiaoman Bi, Qing Zhang, Lei Chen, et al. "}, {"type": "b", "children": [{"type": "t", "text": "NBAS, a gene involved in cytotoxic degranulation, is recurrently mutated in pediatric hemophagocytic lymphohistiocytosis."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "J Hematol Oncol (2022)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1186/s13045-022-01318-z"}], "href": "https://doi.org/10.1186/s13045-022-01318-z"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "35902954"}], "href": "https://pubmed.ncbi.nlm.nih.gov/35902954"}]}]}]}
Synonyms	SOPH
Proteins	NBAS_HUMAN
NCBI Gene ID	51594
API
Download Associations
Predicted Functions
Co-expressed Genes
Expression in Tissues and Cell Lines

Functional Associations

NBAS has 8,522 functional associations with biological entities spanning 9 categories (molecular profile, organism, chemical, disease, phenotype or trait, functional term, phrase or reference, structural feature, cell line, cell type or tissue, gene, protein or microRNA, sequence feature) extracted from 117 datasets.

Click the + buttons to view associations for NBAS from the datasets below.

If available, associations are ranked by standardized value

Harmonizome 3.0

NBAS Gene

Functional Associations

Please acknowledge the Harmonizome in your publications by citing the following reference(s):

	Dataset	Summary
	Allen Brain Atlas Adult Human Brain Tissue Gene Expression Profiles	tissues with high or low expression of NBAS gene relative to other tissues from the Allen Brain Atlas Adult Human Brain Tissue Gene Expression Profiles dataset.
	Allen Brain Atlas Adult Mouse Brain Tissue Gene Expression Profiles	tissues with high or low expression of NBAS gene relative to other tissues from the Allen Brain Atlas Adult Mouse Brain Tissue Gene Expression Profiles dataset.
	Allen Brain Atlas Aging Dementia and Traumatic Brain Injury Tissue Sample Gene Expression Profiles	tissue samples with high or low expression of NBAS gene relative to other tissue samples from the Allen Brain Atlas Aging Dementia and Traumatic Brain Injury Tissue Sample Gene Expression Profiles dataset.
	Allen Brain Atlas Developing Human Brain Tissue Gene Expression Profiles by Microarray	tissue samples with high or low expression of NBAS gene relative to other tissue samples from the Allen Brain Atlas Developing Human Brain Tissue Gene Expression Profiles by Microarray dataset.
	Allen Brain Atlas Developing Human Brain Tissue Gene Expression Profiles by RNA-seq	tissue samples with high or low expression of NBAS gene relative to other tissue samples from the Allen Brain Atlas Developing Human Brain Tissue Gene Expression Profiles by RNA-seq dataset.
	Allen Brain Atlas Prenatal Human Brain Tissue Gene Expression Profiles	tissues with high or low expression of NBAS gene relative to other tissues from the Allen Brain Atlas Prenatal Human Brain Tissue Gene Expression Profiles dataset.
	BioGPS Cell Line Gene Expression Profiles	cell lines with high or low expression of NBAS gene relative to other cell lines from the BioGPS Cell Line Gene Expression Profiles dataset.
	BioGPS Human Cell Type and Tissue Gene Expression Profiles	cell types and tissues with high or low expression of NBAS gene relative to other cell types and tissues from the BioGPS Human Cell Type and Tissue Gene Expression Profiles dataset.
	BioGPS Mouse Cell Type and Tissue Gene Expression Profiles	cell types and tissues with high or low expression of NBAS gene relative to other cell types and tissues from the BioGPS Mouse Cell Type and Tissue Gene Expression Profiles dataset.
	Carcinogenome Chemical Perturbation Carcinogenicity Signatures	small molecule perturbations changing expression of NBAS gene from the Carcinogenome Chemical Perturbation Carcinogenicity Signatures dataset.
	CCLE Cell Line Gene CNV Profiles	cell lines with high or low copy number of NBAS gene relative to other cell lines from the CCLE Cell Line Gene CNV Profiles dataset.
	CCLE Cell Line Gene Expression Profiles	cell lines with high or low expression of NBAS gene relative to other cell lines from the CCLE Cell Line Gene Expression Profiles dataset.
	CCLE Cell Line Proteomics	Cell lines associated with NBAS protein from the CCLE Cell Line Proteomics dataset.
	CellMarker Gene-Cell Type Associations	cell types associated with NBAS gene from the CellMarker Gene-Cell Type Associations dataset.
	ChEA Transcription Factor Binding Site Profiles	transcription factor binding site profiles with transcription factor binding evidence at the promoter of NBAS gene from the CHEA Transcription Factor Binding Site Profiles dataset.
	ChEA Transcription Factor Targets	transcription factors binding the promoter of NBAS gene in low- or high-throughput transcription factor functional studies from the CHEA Transcription Factor Targets dataset.
	ChEA Transcription Factor Targets 2022	transcription factors binding the promoter of NBAS gene in low- or high-throughput transcription factor functional studies from the CHEA Transcription Factor Targets 2022 dataset.
	ClinVar Gene-Phenotype Associations	phenotypes associated with NBAS gene from the curated ClinVar Gene-Phenotype Associations dataset.
	ClinVar Gene-Phenotype Associations 2025	phenotypes associated with NBAS gene from the curated ClinVar Gene-Phenotype Associations 2025 dataset.
	CM4AI U2OS Cell Map Protein Localization Assemblies	assemblies containing NBAS protein from integrated AP-MS and IF data from the CM4AI U2OS Cell Map Protein Localization Assemblies dataset.
	CMAP Signatures of Differentially Expressed Genes for Small Molecules	small molecule perturbations changing expression of NBAS gene from the CMAP Signatures of Differentially Expressed Genes for Small Molecules dataset.
	COMPARTMENTS Curated Protein Localization Evidence Scores	cellular components containing NBAS protein from the COMPARTMENTS Curated Protein Localization Evidence Scores dataset.
	COMPARTMENTS Curated Protein Localization Evidence Scores 2025	cellular components containing NBAS protein from the COMPARTMENTS Curated Protein Localization Evidence Scores 2025 dataset.
	COMPARTMENTS Experimental Protein Localization Evidence Scores 2025	cellular components containing NBAS protein in low- or high-throughput protein localization assays from the COMPARTMENTS Experimental Protein Localization Evidence Scores 2025 dataset.
	COMPARTMENTS Text-mining Protein Localization Evidence Scores	cellular components co-occuring with NBAS protein in abstracts of biomedical publications from the COMPARTMENTS Text-mining Protein Localization Evidence Scores dataset.
	COMPARTMENTS Text-mining Protein Localization Evidence Scores 2025	cellular components co-occuring with NBAS protein in abstracts of biomedical publications from the COMPARTMENTS Text-mining Protein Localization Evidence Scores 2025 dataset.
	COSMIC Cell Line Gene CNV Profiles	cell lines with high or low copy number of NBAS gene relative to other cell lines from the COSMIC Cell Line Gene CNV Profiles dataset.
	COSMIC Cell Line Gene Mutation Profiles	cell lines with NBAS gene mutations from the COSMIC Cell Line Gene Mutation Profiles dataset.
	CTD Gene-Chemical Interactions	chemicals interacting with NBAS gene/protein from the curated CTD Gene-Chemical Interactions dataset.
	CTD Gene-Disease Associations	diseases associated with NBAS gene/protein from the curated CTD Gene-Disease Associations dataset.
	dbGAP Gene-Trait Associations	traits associated with NBAS gene in GWAS and other genetic association datasets from the dbGAP Gene-Trait Associations dataset.
	DISEASES Experimental Gene-Disease Association Evidence Scores	diseases associated with NBAS gene in GWAS datasets from the DISEASES Experimental Gene-Disease Assocation Evidence Scores dataset.
	DISEASES Experimental Gene-Disease Association Evidence Scores 2025	diseases associated with NBAS gene in GWAS datasets from the DISEASES Experimental Gene-Disease Assocation Evidence Scores 2025 dataset.
	DISEASES Text-mining Gene-Disease Association Evidence Scores	diseases co-occuring with NBAS gene in abstracts of biomedical publications from the DISEASES Text-mining Gene-Disease Assocation Evidence Scores dataset.
	DISEASES Text-mining Gene-Disease Association Evidence Scores 2025	diseases co-occuring with NBAS gene in abstracts of biomedical publications from the DISEASES Text-mining Gene-Disease Assocation Evidence Scores 2025 dataset.
	DisGeNET Gene-Disease Associations	diseases associated with NBAS gene in GWAS and other genetic association datasets from the DisGeNET Gene-Disease Associations dataset.
	DisGeNET Gene-Phenotype Associations	phenotypes associated with NBAS gene in GWAS and other genetic association datasets from the DisGeNET Gene-Phenoptype Associations dataset.
	ENCODE Histone Modification Site Profiles	histone modification site profiles with high histone modification abundance at NBAS gene from the ENCODE Histone Modification Site Profiles dataset.
	ENCODE Transcription Factor Binding Site Profiles	transcription factor binding site profiles with transcription factor binding evidence at the promoter of NBAS gene from the ENCODE Transcription Factor Binding Site Profiles dataset.
	ENCODE Transcription Factor Targets	transcription factors binding the promoter of NBAS gene in ChIP-seq datasets from the ENCODE Transcription Factor Targets dataset.