RAD54B Gene

Name	RAD54 homolog B (S. cerevisiae)
Description	The protein encoded by this gene belongs to the DEAD-like helicase superfamily. It shares similarity with Saccharomyces cerevisiae RAD54 and RDH54, both of which are involved in homologous recombination and repair of DNA. This protein binds to double-stranded DNA, and displays ATPase activity in the presence of DNA. This gene is highly expressed in testis and spleen, which suggests active roles in meiotic and mitotic recombination. Homozygous mutations of this gene were observed in primary lymphoma and colon cancer. [provided by RefSeq, Jul 2008]
Summary	{"type": "root", "children": [{"type": "p", "children": [{"type": "t", "text": "\nRAD54B is a DNA-dependent ATPase belonging to the SWI2/SNF2 family that plays a pivotal role in homologous recombination. It has been shown to facilitate DNA repair by modulating recombinase activity – for example, by enhancing the DMC1‐mediated strand exchange through stabilization of the DMC1–single‐stranded DNA complex and by aiding the removal of RAD51 from double‐stranded DNA to prevent the persistence of nonrepair‐associated complexes that could undermine genome stability."}, {"type": "fg", "children": [{"type": "fg_fs", "start_ref": "1", "end_ref": "3"}]}, {"type": "t", "text": ""}]}, {"type": "t", "text": "\n\n"}, {"type": "p", "children": [{"type": "t", "text": "\nBeyond its enzymatic functions, RAD54B contributes to maintaining genomic integrity by influencing cell cycle checkpoints. It has been identified as a scaffold that promotes the degradation of the tumor suppressor p53 via interaction with the MDM2–MDMX complex, thereby limiting checkpoint strength and facilitating cell cycle progression. Moreover, knockdown or mutational inactivation of RAD54B results in impaired double-strand break repair and chromosome instability, leading to synthetic lethal interactions when combined with targeted suppressions of genes such as FEN1 and SOD1."}, {"type": "fg", "children": [{"type": "fg_fs", "start_ref": "4", "end_ref": "6"}]}, {"type": "t", "text": ""}]}, {"type": "t", "text": "\n\n"}, {"type": "p", "children": [{"type": "t", "text": "\nAberrant expression or mutation of RAD54B has been implicated in the oncogenesis of several cancer types, including colorectal, breast, ovarian, liver, and lung cancers. Elevated RAD54B levels or its deregulated function correlate with enhanced cellular proliferation, defective DNA repair, and poor clinical outcomes. These observations substantiate its emerging role as a prognostic biomarker and underscore the potential of exploiting RAD54B-related synthetic lethality as a precision therapeutic strategy in tumors harboring homologous recombination deficiencies."}, {"type": "fg", "children": [{"type": "fg_fs", "start_ref": "7", "end_ref": "12"}]}, {"type": "t", "text": ""}]}, {"type": "t", "text": "\n\n"}, {"type": "p", "children": [{"type": "t", "text": "\nFurthermore, protein–protein interaction studies have revealed that RAD54B is recruited to sites of DNA double-strand break repair through its association with proteins such as S100A11, while biochemical analyses of its N-terminal domain demonstrate its ability to bind branched DNA structures and interact with DMC1, thereby potentially influencing the architecture of recombination complexes."}, {"type": "fg", "children": [{"type": "fg_f", "ref": "13"}]}, {"type": "t", "text": ""}]}, {"type": "t", "text": "\n\n"}, {"type": "p", "children": [{"type": "t", "text": "\nIn addition, the regulation of RAD54B expression itself may be under the control of chromatin-remodeling factors; for instance, the human INO80 complex has been shown to bind the RAD54B promoter and positively regulate its expression, further linking chromatin dynamics with the maintenance of genome stability."}, {"type": "fg", "children": [{"type": "fg_f", "ref": "15"}]}, {"type": "t", "text": ""}]}, {"type": "rg", "children": [{"type": "r", "ref": 1, "children": [{"type": "t", "text": "Kozo Tanaka, Wataru Kagawa, Takashi Kinebuchi, et al. "}, {"type": "b", "children": [{"type": "t", "text": "Human Rad54B is a double-stranded DNA-dependent ATPase and has biochemical properties different from its structural homolog in yeast, Tid1/Rdh54."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Nucleic Acids Res (2002)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1093/nar/30.6.1346"}], "href": "https://doi.org/10.1093/nar/30.6.1346"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "11884632"}], "href": "https://pubmed.ncbi.nlm.nih.gov/11884632"}]}, {"type": "r", "ref": 2, "children": [{"type": "t", "text": "Naoyuki Sarai, Wataru Kagawa, Takashi Kinebuchi, et al. "}, {"type": "b", "children": [{"type": "t", "text": "Stimulation of Dmc1-mediated DNA strand exchange by the human Rad54B protein."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Nucleic Acids Res (2006)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1093/nar/gkl562"}], "href": "https://doi.org/10.1093/nar/gkl562"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "16945962"}], "href": "https://pubmed.ncbi.nlm.nih.gov/16945962"}]}, {"type": "r", "ref": 3, "children": [{"type": "t", "text": "Jennifer M Mason, Kritika Dusad, William Douglass Wright, et al. "}, {"type": "b", "children": [{"type": "t", "text": "RAD54 family translocases counter genotoxic effects of RAD51 in human tumor cells."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Nucleic Acids Res (2015)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1093/nar/gkv175"}], "href": "https://doi.org/10.1093/nar/gkv175"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "25765654"}], "href": "https://pubmed.ncbi.nlm.nih.gov/25765654"}]}, {"type": "r", "ref": 4, "children": [{"type": "t", "text": "Kirk J McManus, Irene J Barrett, Yasaman Nouhi, et al. "}, {"type": "b", "children": [{"type": "t", "text": "Specific synthetic lethal killing of RAD54B-deficient human colorectal cancer cells by FEN1 silencing."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Proc Natl Acad Sci U S A (2009)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1073/pnas.0813414106"}], "href": "https://doi.org/10.1073/pnas.0813414106"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "19218431"}], "href": "https://pubmed.ncbi.nlm.nih.gov/19218431"}]}, {"type": "r", "ref": 5, "children": [{"type": "t", "text": "Babu V Sajesh, Melanie Bailey, Zelda Lichtensztejn, et al. "}, {"type": "b", "children": [{"type": "t", "text": "Synthetic lethal targeting of superoxide dismutase 1 selectively kills RAD54B-deficient colorectal cancer cells."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Genetics (2013)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1534/genetics.113.156836"}], "href": "https://doi.org/10.1534/genetics.113.156836"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "24002644"}], "href": "https://pubmed.ncbi.nlm.nih.gov/24002644"}]}, {"type": "r", "ref": 6, "children": [{"type": "t", "text": "Takaaki Yasuhara, Takahiko Suzuki, Mari Katsura, et al. "}, {"type": "b", "children": [{"type": "t", "text": "Rad54B serves as a scaffold in the DNA damage response that limits checkpoint strength."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Nat Commun (2014)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1038/ncomms6426"}], "href": "https://doi.org/10.1038/ncomms6426"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "25384516"}], "href": "https://pubmed.ncbi.nlm.nih.gov/25384516"}]}, {"type": "r", "ref": 7, "children": [{"type": "t", "text": "Yuzo Nagai, Yoko Yamamoto, Takaaki Yasuhara, et al. "}, {"type": "b", "children": [{"type": "t", "text": "High RAD54B expression: an independent predictor of postoperative distant recurrence in colorectal cancer patients."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Oncotarget (2015)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.18632/oncotarget.4222"}], "href": "https://doi.org/10.18632/oncotarget.4222"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "26046797"}], "href": "https://pubmed.ncbi.nlm.nih.gov/26046797"}]}, {"type": "r", "ref": 8, "children": [{"type": "t", "text": "Jau-Chung Hwang, Wen-Wei Sung, Hung-Pin Tu, et al. "}, {"type": "b", "children": [{"type": "t", "text": "The Overexpression of FEN1 and RAD54B May Act as Independent Prognostic Factors of Lung Adenocarcinoma."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "PLoS One (2015)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1371/journal.pone.0139435"}], "href": "https://doi.org/10.1371/journal.pone.0139435"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "26431531"}], "href": "https://pubmed.ncbi.nlm.nih.gov/26431531"}]}, {"type": "r", "ref": 9, "children": [{"type": "t", "text": "Rui Wang, Yawei Li, Yufo Chen, et al. "}, {"type": "b", "children": [{"type": "t", "text": "Inhibition of RAD54B suppresses proliferation and promotes apoptosis in hepatoma cells."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Oncol Rep (2018)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.3892/or.2018.6522"}], "href": "https://doi.org/10.3892/or.2018.6522"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "29956808"}], "href": "https://pubmed.ncbi.nlm.nih.gov/29956808"}]}, {"type": "r", "ref": 10, "children": [{"type": "t", "text": "Zeyu Zhang, Xiaoting Li, Yingyan Han, et al. "}, {"type": "b", "children": [{"type": "t", "text": "RAD54B potentiates tumor growth and predicts poor prognosis of patients with luminal A breast cancer."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Biomed Pharmacother (2019)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1016/j.biopha.2019.109341"}], "href": "https://doi.org/10.1016/j.biopha.2019.109341"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "31545289"}], "href": "https://pubmed.ncbi.nlm.nih.gov/31545289"}]}, {"type": "r", "ref": 11, "children": [{"type": "t", "text": "Chuan Xu, Di Liu, Hong Mei, et al. "}, {"type": "b", "children": [{"type": "t", "text": "Knockdown of RAD54B expression reduces cell proliferation and induces apoptosis in lung cancer cells."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "J Int Med Res (2019)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1177/0300060519869423"}], "href": "https://doi.org/10.1177/0300060519869423"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "31558081"}], "href": "https://pubmed.ncbi.nlm.nih.gov/31558081"}]}, {"type": "r", "ref": 12, "children": [{"type": "t", "text": "Peng Liu, Chunxiu Lin, Lanlan Liu, et al. "}, {"type": "b", "children": [{"type": "t", "text": "RAD54B mutations enhance the sensitivity of ovarian cancer cells to poly(ADP-ribose) polymerase (PARP) inhibitors."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "J Biol Chem (2022)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1016/j.jbc.2022.102354"}], "href": "https://doi.org/10.1016/j.jbc.2022.102354"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "35952757"}], "href": "https://pubmed.ncbi.nlm.nih.gov/35952757"}]}, {"type": "r", "ref": 13, "children": [{"type": "t", "text": "Ulrike Murzik, Peter Hemmerich, Stefanie Weidtkamp-Peters, et al. "}, {"type": "b", "children": [{"type": "t", "text": "Rad54B targeting to DNA double-strand break repair sites requires complex formation with S100A11."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Mol Biol Cell (2008)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1091/mbc.e07-11-1167"}], "href": "https://doi.org/10.1091/mbc.e07-11-1167"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "18463164"}], "href": "https://pubmed.ncbi.nlm.nih.gov/18463164"}]}, {"type": "r", "ref": 14, "children": [{"type": "t", "text": "Naoyuki Sarai, Wataru Kagawa, Norie Fujikawa, et al. "}, {"type": "b", "children": [{"type": "t", "text": "Biochemical analysis of the N-terminal domain of human RAD54B."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Nucleic Acids Res (2008)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1093/nar/gkn516"}], "href": "https://doi.org/10.1093/nar/gkn516"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "18718930"}], "href": "https://pubmed.ncbi.nlm.nih.gov/18718930"}]}, {"type": "r", "ref": 15, "children": [{"type": "t", "text": "Eun-Jung Park, Shin-Kyoung Hur, Jongbum Kwon "}, {"type": "b", "children": [{"type": "t", "text": "Human INO80 chromatin-remodelling complex contributes to DNA double-strand break repair via the expression of Rad54B and XRCC3 genes."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Biochem J (2010)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1042/BJ20100988"}], "href": "https://doi.org/10.1042/BJ20100988"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "20687897"}], "href": "https://pubmed.ncbi.nlm.nih.gov/20687897"}]}]}]}
Synonyms	RDH54
Proteins	RA54B_HUMAN
NCBI Gene ID	25788
API
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Predicted Functions
Co-expressed Genes
Expression in Tissues and Cell Lines

Functional Associations

RAD54B has 6,053 functional associations with biological entities spanning 8 categories (molecular profile, organism, chemical, functional term, phrase or reference, disease, phenotype or trait, structural feature, cell line, cell type or tissue, gene, protein or microRNA) extracted from 116 datasets.

Click the + buttons to view associations for RAD54B from the datasets below.

If available, associations are ranked by standardized value

Harmonizome 3.0

RAD54B Gene

Functional Associations

Please acknowledge the Harmonizome in your publications by citing the following reference(s):

	Dataset	Summary
	Allen Brain Atlas Adult Human Brain Tissue Gene Expression Profiles	tissues with high or low expression of RAD54B gene relative to other tissues from the Allen Brain Atlas Adult Human Brain Tissue Gene Expression Profiles dataset.
	Allen Brain Atlas Adult Mouse Brain Tissue Gene Expression Profiles	tissues with high or low expression of RAD54B gene relative to other tissues from the Allen Brain Atlas Adult Mouse Brain Tissue Gene Expression Profiles dataset.
	Allen Brain Atlas Aging Dementia and Traumatic Brain Injury Tissue Sample Gene Expression Profiles	tissue samples with high or low expression of RAD54B gene relative to other tissue samples from the Allen Brain Atlas Aging Dementia and Traumatic Brain Injury Tissue Sample Gene Expression Profiles dataset.
	Allen Brain Atlas Developing Human Brain Tissue Gene Expression Profiles by Microarray	tissue samples with high or low expression of RAD54B gene relative to other tissue samples from the Allen Brain Atlas Developing Human Brain Tissue Gene Expression Profiles by Microarray dataset.
	Allen Brain Atlas Developing Human Brain Tissue Gene Expression Profiles by RNA-seq	tissue samples with high or low expression of RAD54B gene relative to other tissue samples from the Allen Brain Atlas Developing Human Brain Tissue Gene Expression Profiles by RNA-seq dataset.
	Allen Brain Atlas Prenatal Human Brain Tissue Gene Expression Profiles	tissues with high or low expression of RAD54B gene relative to other tissues from the Allen Brain Atlas Prenatal Human Brain Tissue Gene Expression Profiles dataset.
	BioGPS Cell Line Gene Expression Profiles	cell lines with high or low expression of RAD54B gene relative to other cell lines from the BioGPS Cell Line Gene Expression Profiles dataset.
	BioGPS Human Cell Type and Tissue Gene Expression Profiles	cell types and tissues with high or low expression of RAD54B gene relative to other cell types and tissues from the BioGPS Human Cell Type and Tissue Gene Expression Profiles dataset.
	BioGPS Mouse Cell Type and Tissue Gene Expression Profiles	cell types and tissues with high or low expression of RAD54B gene relative to other cell types and tissues from the BioGPS Mouse Cell Type and Tissue Gene Expression Profiles dataset.
	Carcinogenome Chemical Perturbation Carcinogenicity Signatures	small molecule perturbations changing expression of RAD54B gene from the Carcinogenome Chemical Perturbation Carcinogenicity Signatures dataset.
	CCLE Cell Line Gene CNV Profiles	cell lines with high or low copy number of RAD54B gene relative to other cell lines from the CCLE Cell Line Gene CNV Profiles dataset.
	CCLE Cell Line Gene Expression Profiles	cell lines with high or low expression of RAD54B gene relative to other cell lines from the CCLE Cell Line Gene Expression Profiles dataset.
	CCLE Cell Line Proteomics	Cell lines associated with RAD54B protein from the CCLE Cell Line Proteomics dataset.
	CellMarker Gene-Cell Type Associations	cell types associated with RAD54B gene from the CellMarker Gene-Cell Type Associations dataset.
	ChEA Transcription Factor Binding Site Profiles	transcription factor binding site profiles with transcription factor binding evidence at the promoter of RAD54B gene from the CHEA Transcription Factor Binding Site Profiles dataset.
	ChEA Transcription Factor Targets	transcription factors binding the promoter of RAD54B gene in low- or high-throughput transcription factor functional studies from the CHEA Transcription Factor Targets dataset.
	ChEA Transcription Factor Targets 2022	transcription factors binding the promoter of RAD54B gene in low- or high-throughput transcription factor functional studies from the CHEA Transcription Factor Targets 2022 dataset.
	CMAP Signatures of Differentially Expressed Genes for Small Molecules	small molecule perturbations changing expression of RAD54B gene from the CMAP Signatures of Differentially Expressed Genes for Small Molecules dataset.
	COMPARTMENTS Curated Protein Localization Evidence Scores	cellular components containing RAD54B protein from the COMPARTMENTS Curated Protein Localization Evidence Scores dataset.
	COMPARTMENTS Text-mining Protein Localization Evidence Scores	cellular components co-occuring with RAD54B protein in abstracts of biomedical publications from the COMPARTMENTS Text-mining Protein Localization Evidence Scores dataset.
	COMPARTMENTS Text-mining Protein Localization Evidence Scores 2025	cellular components co-occuring with RAD54B protein in abstracts of biomedical publications from the COMPARTMENTS Text-mining Protein Localization Evidence Scores 2025 dataset.
	COSMIC Cell Line Gene CNV Profiles	cell lines with high or low copy number of RAD54B gene relative to other cell lines from the COSMIC Cell Line Gene CNV Profiles dataset.
	COSMIC Cell Line Gene Mutation Profiles	cell lines with RAD54B gene mutations from the COSMIC Cell Line Gene Mutation Profiles dataset.
	CTD Gene-Chemical Interactions	chemicals interacting with RAD54B gene/protein from the curated CTD Gene-Chemical Interactions dataset.
	CTD Gene-Disease Associations	diseases associated with RAD54B gene/protein from the curated CTD Gene-Disease Associations dataset.
	dbGAP Gene-Trait Associations	traits associated with RAD54B gene in GWAS and other genetic association datasets from the dbGAP Gene-Trait Associations dataset.
	DeepCoverMOA Drug Mechanisms of Action	small molecule perturbations with high or low expression of RAD54B protein relative to other small molecule perturbations from the DeepCoverMOA Drug Mechanisms of Action dataset.
	DepMap CRISPR Gene Dependency	cell lines with fitness changed by RAD54B gene knockdown relative to other cell lines from the DepMap CRISPR Gene Dependency dataset.
	DISEASES Experimental Gene-Disease Association Evidence Scores 2025	diseases associated with RAD54B gene in GWAS datasets from the DISEASES Experimental Gene-Disease Assocation Evidence Scores 2025 dataset.
	DISEASES Text-mining Gene-Disease Association Evidence Scores	diseases co-occuring with RAD54B gene in abstracts of biomedical publications from the DISEASES Text-mining Gene-Disease Assocation Evidence Scores dataset.
	DISEASES Text-mining Gene-Disease Association Evidence Scores 2025	diseases co-occuring with RAD54B gene in abstracts of biomedical publications from the DISEASES Text-mining Gene-Disease Assocation Evidence Scores 2025 dataset.
	DisGeNET Gene-Disease Associations	diseases associated with RAD54B gene in GWAS and other genetic association datasets from the DisGeNET Gene-Disease Associations dataset.
	DisGeNET Gene-Phenotype Associations	phenotypes associated with RAD54B gene in GWAS and other genetic association datasets from the DisGeNET Gene-Phenoptype Associations dataset.
	ENCODE Histone Modification Site Profiles	histone modification site profiles with high histone modification abundance at RAD54B gene from the ENCODE Histone Modification Site Profiles dataset.
	ENCODE Transcription Factor Binding Site Profiles	transcription factor binding site profiles with transcription factor binding evidence at the promoter of RAD54B gene from the ENCODE Transcription Factor Binding Site Profiles dataset.
	ENCODE Transcription Factor Targets	transcription factors binding the promoter of RAD54B gene in ChIP-seq datasets from the ENCODE Transcription Factor Targets dataset.
	ESCAPE Omics Signatures of Genes and Proteins for Stem Cells	PubMedIDs of publications reporting gene signatures containing RAD54B from the ESCAPE Omics Signatures of Genes and Proteins for Stem Cells dataset.
	GAD Gene-Disease Associations	diseases associated with RAD54B gene in GWAS and other genetic association datasets from the GAD Gene-Disease Associations dataset.
	GAD High Level Gene-Disease Associations	diseases associated with RAD54B gene in GWAS and other genetic association datasets from the GAD High Level Gene-Disease Associations dataset.
	GDSC Cell Line Gene Expression Profiles	cell lines with high or low expression of RAD54B gene relative to other cell lines from the GDSC Cell Line Gene Expression Profiles dataset.