ZMYND11 Gene

HGNC Family	Zinc fingers
Name	zinc finger, MYND-type containing 11
Description	The protein encoded by this gene was first identified by its ability to bind the adenovirus E1A protein. The protein localizes to the nucleus. It functions as a transcriptional repressor, and expression of E1A inhibits this repression. Alternatively spliced transcript variants encoding different isoforms have been identified. [provided by RefSeq, Jul 2008]
Summary	{"type": "root", "children": [{"type": "p", "children": [{"type": "t", "text": "\nZMYND11 functions as a highly specific chromatin “reader” that binds the trimethylated lysine‐36 mark on the histone variant H3.3. Structural studies reveal that its tandem bromo‐PWWP domains not only form an aromatic cage for engaging H3K36me3 but also create a composite pocket that accommodates an H3.3‐specific residue, thereby modulating RNA polymerase II elongation and repressing cryptic transcription. This epigenetic “gatekeeper” role ensures proper transcriptional fidelity by preventing aberrant elongation events."}, {"type": "fg", "children": [{"type": "fg_f", "ref": "1"}]}, {"type": "t", "text": ""}]}, {"type": "t", "text": "\n\n"}, {"type": "p", "children": [{"type": "t", "text": "\nBeyond its canonical role in transcriptional elongation control, ZMYND11 also links chromatin status to pre‐mRNA processing. It associates with key RNA splicing regulators to promote regulated intron retention—a less understood alternative splicing event—thereby fine‐tuning gene expression programs. In addition, oncogenic rearrangements that fuse ZMYND11 to MBTD1 in subsets of acute myeloid leukemia, as well as other genetic alterations, underscore its critical involvement in suppressing pro‐leukemic transcriptional circuits."}, {"type": "fg", "children": [{"type": "fg_fs", "start_ref": "3", "end_ref": "6"}]}, {"type": "t", "text": ""}]}, {"type": "t", "text": "\n\n"}, {"type": "p", "children": [{"type": "t", "text": "\nMoreover, ZMYND11 operates as a multifaceted tumor suppressor and developmental regulator. Reduced expression or loss‐of‐function variants are linked to aggressive cancers such as breast cancer, glioblastoma, and hematologic malignancies, as well as neurodevelopmental syndromes characterized by intellectual disability, seizures, and distinct dysmorphic features. In diverse cellular contexts its activity is further modulated via direct interactions that attenuate oncogenic transcription factors (for example, tempering ETS2‐mediated gene activation) and through binding to viral oncoproteins—including adenoviral E1A and Epstein–Barr virus EBNA2—to influence signaling cascades such as NF‐κB activation and p53–p21–dependent senescence. Collectively, these observations highlight how ZMYND11 integrates chromatin–based epigenetic control with RNA processing, transcriptional repression, and signal modulation to maintain cellular homeostasis."}, {"type": "fg", "children": [{"type": "fg_fs", "start_ref": "7", "end_ref": "23"}]}, {"type": "t", "text": ""}]}, {"type": "rg", "children": [{"type": "r", "ref": 1, "children": [{"type": "t", "text": "Hong Wen, Yuanyuan Li, Yuanxin Xi, et al. "}, {"type": "b", "children": [{"type": "t", "text": "ZMYND11 links histone H3.3K36me3 to transcription elongation and tumour suppression."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Nature (2014)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1038/nature13045"}], "href": "https://doi.org/10.1038/nature13045"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "24590075"}], "href": "https://pubmed.ncbi.nlm.nih.gov/24590075"}]}, {"type": "r", "ref": 2, "children": [{"type": "t", "text": "Unknown Authors "}, {"type": "b", "children": [{"type": "t", "text": "The putative tumor suppressor ZMYND11 recognizes H3.3K36me3."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Cancer Discov (2014)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1158/2159-8290.CD-RW2014-065"}], "href": "https://doi.org/10.1158/2159-8290.CD-RW2014-065"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "24795016"}], "href": "https://pubmed.ncbi.nlm.nih.gov/24795016"}]}, {"type": "r", "ref": 3, "children": [{"type": "t", "text": "Rui Guo, Lijuan Zheng, Juw Won Park, et al. "}, {"type": "b", "children": [{"type": "t", "text": "BS69/ZMYND11 reads and connects histone H3.3 lysine 36 trimethylation-decorated chromatin to regulated pre-mRNA processing."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Mol Cell (2014)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1016/j.molcel.2014.08.022"}], "href": "https://doi.org/10.1016/j.molcel.2014.08.022"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "25263594"}], "href": "https://pubmed.ncbi.nlm.nih.gov/25263594"}]}, {"type": "r", "ref": 4, "children": [{"type": "t", "text": "Jie Li, Phillip M Galbo, Weida Gong, et al. "}, {"type": "b", "children": [{"type": "t", "text": "ZMYND11-MBTD1 induces leukemogenesis through hijacking NuA4/TIP60 acetyltransferase complex and a PWWP-mediated chromatin association mechanism."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Nat Commun (2021)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1038/s41467-021-21357-3"}], "href": "https://doi.org/10.1038/s41467-021-21357-3"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "33594072"}], "href": "https://pubmed.ncbi.nlm.nih.gov/33594072"}]}, {"type": "r", "ref": 5, "children": [{"type": "t", "text": "Maëva Devoucoux, Victoire Fort, Gabriel Khelifi, et al. "}, {"type": "b", "children": [{"type": "t", "text": "Oncogenic ZMYND11-MBTD1 fusion protein anchors the NuA4/TIP60 histone acetyltransferase complex to the coding region of active genes."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Cell Rep (2022)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1016/j.celrep.2022.110947"}], "href": "https://doi.org/10.1016/j.celrep.2022.110947"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "35705031"}], "href": "https://pubmed.ncbi.nlm.nih.gov/35705031"}]}, {"type": "r", "ref": 6, "children": [{"type": "t", "text": "Jasmijn D E de Rooij, Marry M van den Heuvel-Eibrink, Wouter J W Kollen, et al. "}, {"type": "b", "children": [{"type": "t", "text": "Recurrent translocation t(10;17)(p15;q21) in minimally differentiated acute myeloid leukemia results in ZMYND11/MBTD1 fusion."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Genes Chromosomes Cancer (2016)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1002/gcc.22326"}], "href": "https://doi.org/10.1002/gcc.22326"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "26608508"}], "href": "https://pubmed.ncbi.nlm.nih.gov/26608508"}]}, {"type": "r", "ref": 7, "children": [{"type": "t", "text": "Joshua P Plotnik, Peter C Hollenhorst "}, {"type": "b", "children": [{"type": "t", "text": "Interaction with ZMYND11 mediates opposing roles of Ras-responsive transcription factors ETS1 and ETS2."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Nucleic Acids Res (2017)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1093/nar/gkx039"}], "href": "https://doi.org/10.1093/nar/gkx039"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "28119415"}], "href": "https://pubmed.ncbi.nlm.nih.gov/28119415"}]}, {"type": "r", "ref": 8, "children": [{"type": "t", "text": "Birute Tumiene, Ž Čiuladaitė, E Preikšaitienė, et al. "}, {"type": "b", "children": [{"type": "t", "text": "Phenotype comparison confirms ZMYND11 as a critical gene for 10p15.3 microdeletion syndrome."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "J Appl Genet (2017)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1007/s13353-017-0408-3"}], "href": "https://doi.org/10.1007/s13353-017-0408-3"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "28933030"}], "href": "https://pubmed.ncbi.nlm.nih.gov/28933030"}]}, {"type": "r", "ref": 9, "children": [{"type": "t", "text": "Ji-Peng Yang, Jian-Kai Yang, Chen Li, et al. "}, {"type": "b", "children": [{"type": "t", "text": "Downregulation of ZMYND11 induced by miR-196a-5p promotes the progression and growth of GBM."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Biochem Biophys Res Commun (2017)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1016/j.bbrc.2017.10.098"}], "href": "https://doi.org/10.1016/j.bbrc.2017.10.098"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "29066350"}], "href": "https://pubmed.ncbi.nlm.nih.gov/29066350"}]}, {"type": "r", "ref": 10, "children": [{"type": "t", "text": "Thabo M Yates, Morgan Drucker, Angela Barnicoat, et al. "}, {"type": "b", "children": [{"type": "t", "text": "ZMYND11-related syndromic intellectual disability: 16 patients delineating and expanding the phenotypic spectrum."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Hum Mutat (2020)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1002/humu.24001"}], "href": "https://doi.org/10.1002/humu.24001"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "32097528"}], "href": "https://pubmed.ncbi.nlm.nih.gov/32097528"}]}, {"type": "r", "ref": 11, "children": [{"type": "t", "text": "Stéphane Ansieau, Achim Leutz "}, {"type": "b", "children": [{"type": "t", "text": "The conserved Mynd domain of BS69 binds cellular and oncoviral proteins through a common PXLXP motif."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "J Biol Chem (2002)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1074/jbc.M110078200"}], "href": "https://doi.org/10.1074/jbc.M110078200"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "11733528"}], "href": "https://pubmed.ncbi.nlm.nih.gov/11733528"}]}, {"type": "r", "ref": 12, "children": [{"type": "t", "text": "H Masselink, R Bernards "}, {"type": "b", "children": [{"type": "t", "text": "The adenovirus E1A binding protein BS69 is a corepressor of transcription through recruitment of N-CoR."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Oncogene (2000)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1038/sj.onc.1203421"}], "href": "https://doi.org/10.1038/sj.onc.1203421"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "10734313"}], "href": "https://pubmed.ncbi.nlm.nih.gov/10734313"}]}, {"type": "r", "ref": 13, "children": [{"type": "t", "text": "Jun Wan, Wei Zhang, Liming Wu, et al. "}, {"type": "b", "children": [{"type": "t", "text": "BS69, a specific adaptor in the latent membrane protein 1-mediated c-Jun N-terminal kinase pathway."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Mol Cell Biol (2006)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1128/MCB.26.2.448-456.2006"}], "href": "https://doi.org/10.1128/MCB.26.2.448-456.2006"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "16382137"}], "href": "https://pubmed.ncbi.nlm.nih.gov/16382137"}]}, {"type": "r", "ref": 14, "children": [{"type": "t", "text": "Guillaume Velasco, Steve Grkovic, Stéphane Ansieau "}, {"type": "b", "children": [{"type": "t", "text": "New insights into BS69 functions."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "J Biol Chem (2006)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1074/jbc.M600573200"}], "href": "https://doi.org/10.1074/jbc.M600573200"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "16565076"}], "href": "https://pubmed.ncbi.nlm.nih.gov/16565076"}]}, {"type": "r", "ref": 15, "children": [{"type": "t", "text": "Rajesh Ponnusamy, Ritika Khatri, Paulo B Correia, et al. "}, {"type": "b", "children": [{"type": "t", "text": "Increased association between Epstein-Barr virus EBNA2 from type 2 strains and the transcriptional repressor BS69 restricts EBNA2 activity."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "PLoS Pathog (2019)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1371/journal.ppat.1007458"}], "href": "https://doi.org/10.1371/journal.ppat.1007458"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "31283782"}], "href": "https://pubmed.ncbi.nlm.nih.gov/31283782"}]}, {"type": "r", "ref": 16, "children": [{"type": "t", "text": "Osamu Ikeda, Yuto Miyasaka, Ryuji Yoshida, et al. "}, {"type": "b", "children": [{"type": "t", "text": "BS69 cooperates with TRAF3 in the regulation of Epstein-Barr virus-derived LMP1/CTAR1-induced NF-kappaB activation."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "FEBS Lett (2010)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1016/j.febslet.2010.01.060"}], "href": "https://doi.org/10.1016/j.febslet.2010.01.060"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "20138174"}], "href": "https://pubmed.ncbi.nlm.nih.gov/20138174"}]}, {"type": "r", "ref": 17, "children": [{"type": "t", "text": "Bo Yu, Yong Shao, Chao Zhang, et al. "}, {"type": "b", "children": [{"type": "t", "text": "BS69 undergoes SUMO modification and plays an inhibitory role in muscle and neuronal differentiation."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Exp Cell Res (2009)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1016/j.yexcr.2009.09.011"}], "href": "https://doi.org/10.1016/j.yexcr.2009.09.011"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "19766626"}], "href": "https://pubmed.ncbi.nlm.nih.gov/19766626"}]}, {"type": "r", "ref": 18, "children": [{"type": "t", "text": "Tomoyasu Isobe, Chiharu Uchida, Takayuki Hattori, et al. "}, {"type": "b", "children": [{"type": "t", "text": "Ubiquitin-dependent degradation of adenovirus E1A protein is inhibited by BS69."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Biochem Biophys Res Commun (2006)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1016/j.bbrc.2005.11.028"}], "href": "https://doi.org/10.1016/j.bbrc.2005.11.028"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "16300738"}], "href": "https://pubmed.ncbi.nlm.nih.gov/16300738"}]}, {"type": "r", "ref": 19, "children": [{"type": "t", "text": "Hiromi Takaki, Hiroyuki Oshiumi, Miwa Sasai, et al. "}, {"type": "b", "children": [{"type": "t", "text": "Oligomerized TICAM-1 (TRIF) in the cytoplasm recruits nuclear BS69 to enhance NF-kappaB activation and type I IFN induction."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Eur J Immunol (2009)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1002/eji.200939878"}], "href": "https://doi.org/10.1002/eji.200939878"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "19795416"}], "href": "https://pubmed.ncbi.nlm.nih.gov/19795416"}]}, {"type": "r", "ref": 20, "children": [{"type": "t", "text": "Hong Yang, Chao Zhang, Xiaosu Zhao, et al. "}, {"type": "b", "children": [{"type": "t", "text": "Analysis of copy number variations of BS69 in multiple types of hematological malignancies."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Ann Hematol (2010)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1007/s00277-010-0966-5"}], "href": "https://doi.org/10.1007/s00277-010-0966-5"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "20425112"}], "href": "https://pubmed.ncbi.nlm.nih.gov/20425112"}]}, {"type": "r", "ref": 21, "children": [{"type": "t", "text": "Ali Zhang, Tanner M Tessier, Kristianne J C Galpin, et al. "}, {"type": "b", "children": [{"type": "t", "text": "The Transcriptional Repressor BS69 is a Conserved Target of the E1A Proteins from Several Human Adenovirus Species."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Viruses (2018)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.3390/v10120662"}], "href": "https://doi.org/10.3390/v10120662"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "30469473"}], "href": "https://pubmed.ncbi.nlm.nih.gov/30469473"}]}, {"type": "r", "ref": 22, "children": [{"type": "t", "text": "Hao-Ping Liu, Pei-Jung Chung, Chih-Lung Liang, et al. "}, {"type": "b", "children": [{"type": "t", "text": "The MYND domain-containing protein BRAM1 inhibits lymphotoxin beta receptor-mediated signaling through affecting receptor oligomerization."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Cell Signal (2011)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1016/j.cellsig.2010.08.006"}], "href": "https://doi.org/10.1016/j.cellsig.2010.08.006"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "20732415"}], "href": "https://pubmed.ncbi.nlm.nih.gov/20732415"}]}, {"type": "r", "ref": 23, "children": [{"type": "t", "text": "Stephanie Oates, Michael Absoud, Sushma Goyal, et al. "}, {"type": "b", "children": [{"type": "t", "text": "ZMYND11 variants are a novel cause of centrotemporal and generalised epilepsies with neurodevelopmental disorder."}]}, {"type": "t", "text": " "}, {"type": "i", "children": [{"type": "t", "text": "Clin Genet (2021)"}]}, {"type": "t", "text": " DOI: "}, {"type": "a", "children": [{"type": "t", "text": "10.1111/cge.14023"}], "href": "https://doi.org/10.1111/cge.14023"}, {"type": "t", "text": " PMID: "}, {"type": "a", "children": [{"type": "t", "text": "34216016"}], "href": "https://pubmed.ncbi.nlm.nih.gov/34216016"}]}]}]}
Synonyms	BRAM1, BS69, MRD30
Proteins	ZMY11_HUMAN
NCBI Gene ID	10771
API
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Predicted Functions
Co-expressed Genes
Expression in Tissues and Cell Lines

Functional Associations

ZMYND11 has 6,653 functional associations with biological entities spanning 8 categories (molecular profile, organism, disease, phenotype or trait, chemical, functional term, phrase or reference, structural feature, cell line, cell type or tissue, gene, protein or microRNA) extracted from 124 datasets.

Click the + buttons to view associations for ZMYND11 from the datasets below.

If available, associations are ranked by standardized value

Harmonizome 3.0

ZMYND11 Gene

Functional Associations

Please acknowledge the Harmonizome in your publications by citing the following reference(s):

	Dataset	Summary
	Achilles Cell Line Gene Essentiality Profiles	cell lines with fitness changed by ZMYND11 gene knockdown relative to other cell lines from the Achilles Cell Line Gene Essentiality Profiles dataset.
	Allen Brain Atlas Adult Human Brain Tissue Gene Expression Profiles	tissues with high or low expression of ZMYND11 gene relative to other tissues from the Allen Brain Atlas Adult Human Brain Tissue Gene Expression Profiles dataset.
	Allen Brain Atlas Adult Mouse Brain Tissue Gene Expression Profiles	tissues with high or low expression of ZMYND11 gene relative to other tissues from the Allen Brain Atlas Adult Mouse Brain Tissue Gene Expression Profiles dataset.
	Allen Brain Atlas Aging Dementia and Traumatic Brain Injury Tissue Sample Gene Expression Profiles	tissue samples with high or low expression of ZMYND11 gene relative to other tissue samples from the Allen Brain Atlas Aging Dementia and Traumatic Brain Injury Tissue Sample Gene Expression Profiles dataset.
	Allen Brain Atlas Developing Human Brain Tissue Gene Expression Profiles by Microarray	tissue samples with high or low expression of ZMYND11 gene relative to other tissue samples from the Allen Brain Atlas Developing Human Brain Tissue Gene Expression Profiles by Microarray dataset.
	Allen Brain Atlas Developing Human Brain Tissue Gene Expression Profiles by RNA-seq	tissue samples with high or low expression of ZMYND11 gene relative to other tissue samples from the Allen Brain Atlas Developing Human Brain Tissue Gene Expression Profiles by RNA-seq dataset.
	Allen Brain Atlas Prenatal Human Brain Tissue Gene Expression Profiles	tissues with high or low expression of ZMYND11 gene relative to other tissues from the Allen Brain Atlas Prenatal Human Brain Tissue Gene Expression Profiles dataset.
	BioGPS Cell Line Gene Expression Profiles	cell lines with high or low expression of ZMYND11 gene relative to other cell lines from the BioGPS Cell Line Gene Expression Profiles dataset.
	BioGPS Human Cell Type and Tissue Gene Expression Profiles	cell types and tissues with high or low expression of ZMYND11 gene relative to other cell types and tissues from the BioGPS Human Cell Type and Tissue Gene Expression Profiles dataset.
	BioGPS Mouse Cell Type and Tissue Gene Expression Profiles	cell types and tissues with high or low expression of ZMYND11 gene relative to other cell types and tissues from the BioGPS Mouse Cell Type and Tissue Gene Expression Profiles dataset.
	CCLE Cell Line Gene CNV Profiles	cell lines with high or low copy number of ZMYND11 gene relative to other cell lines from the CCLE Cell Line Gene CNV Profiles dataset.
	CCLE Cell Line Gene Expression Profiles	cell lines with high or low expression of ZMYND11 gene relative to other cell lines from the CCLE Cell Line Gene Expression Profiles dataset.
	CCLE Cell Line Proteomics	Cell lines associated with ZMYND11 protein from the CCLE Cell Line Proteomics dataset.
	CellMarker Gene-Cell Type Associations	cell types associated with ZMYND11 gene from the CellMarker Gene-Cell Type Associations dataset.
	ChEA Transcription Factor Binding Site Profiles	transcription factor binding site profiles with transcription factor binding evidence at the promoter of ZMYND11 gene from the CHEA Transcription Factor Binding Site Profiles dataset.
	ChEA Transcription Factor Targets	transcription factors binding the promoter of ZMYND11 gene in low- or high-throughput transcription factor functional studies from the CHEA Transcription Factor Targets dataset.
	ChEA Transcription Factor Targets 2022	transcription factors binding the promoter of ZMYND11 gene in low- or high-throughput transcription factor functional studies from the CHEA Transcription Factor Targets 2022 dataset.
	ClinVar Gene-Phenotype Associations	phenotypes associated with ZMYND11 gene from the curated ClinVar Gene-Phenotype Associations dataset.
	ClinVar Gene-Phenotype Associations 2025	phenotypes associated with ZMYND11 gene from the curated ClinVar Gene-Phenotype Associations 2025 dataset.
	CMAP Signatures of Differentially Expressed Genes for Small Molecules	small molecule perturbations changing expression of ZMYND11 gene from the CMAP Signatures of Differentially Expressed Genes for Small Molecules dataset.
	COMPARTMENTS Curated Protein Localization Evidence Scores	cellular components containing ZMYND11 protein from the COMPARTMENTS Curated Protein Localization Evidence Scores dataset.
	COMPARTMENTS Experimental Protein Localization Evidence Scores	cellular components containing ZMYND11 protein in low- or high-throughput protein localization assays from the COMPARTMENTS Experimental Protein Localization Evidence Scores dataset.
	COMPARTMENTS Text-mining Protein Localization Evidence Scores	cellular components co-occuring with ZMYND11 protein in abstracts of biomedical publications from the COMPARTMENTS Text-mining Protein Localization Evidence Scores dataset.
	COMPARTMENTS Text-mining Protein Localization Evidence Scores 2025	cellular components co-occuring with ZMYND11 protein in abstracts of biomedical publications from the COMPARTMENTS Text-mining Protein Localization Evidence Scores 2025 dataset.
	COSMIC Cell Line Gene CNV Profiles	cell lines with high or low copy number of ZMYND11 gene relative to other cell lines from the COSMIC Cell Line Gene CNV Profiles dataset.
	COSMIC Cell Line Gene Mutation Profiles	cell lines with ZMYND11 gene mutations from the COSMIC Cell Line Gene Mutation Profiles dataset.
	CTD Gene-Disease Associations	diseases associated with ZMYND11 gene/protein from the curated CTD Gene-Disease Associations dataset.
	dbGAP Gene-Trait Associations	traits associated with ZMYND11 gene in GWAS and other genetic association datasets from the dbGAP Gene-Trait Associations dataset.
	DeepCoverMOA Drug Mechanisms of Action	small molecule perturbations with high or low expression of ZMYND11 protein relative to other small molecule perturbations from the DeepCoverMOA Drug Mechanisms of Action dataset.
	DepMap CRISPR Gene Dependency	cell lines with fitness changed by ZMYND11 gene knockdown relative to other cell lines from the DepMap CRISPR Gene Dependency dataset.
	DISEASES Curated Gene-Disease Association Evidence Scores	diseases involving ZMYND11 gene from the DISEASES Curated Gene-Disease Assocation Evidence Scores dataset.
	DISEASES Curated Gene-Disease Association Evidence Scores 2025	diseases involving ZMYND11 gene from the DISEASES Curated Gene-Disease Association Evidence Scores 2025 dataset.
	DISEASES Text-mining Gene-Disease Association Evidence Scores	diseases co-occuring with ZMYND11 gene in abstracts of biomedical publications from the DISEASES Text-mining Gene-Disease Assocation Evidence Scores dataset.
	DISEASES Text-mining Gene-Disease Association Evidence Scores 2025	diseases co-occuring with ZMYND11 gene in abstracts of biomedical publications from the DISEASES Text-mining Gene-Disease Assocation Evidence Scores 2025 dataset.
	DisGeNET Gene-Disease Associations	diseases associated with ZMYND11 gene in GWAS and other genetic association datasets from the DisGeNET Gene-Disease Associations dataset.
	DisGeNET Gene-Phenotype Associations	phenotypes associated with ZMYND11 gene in GWAS and other genetic association datasets from the DisGeNET Gene-Phenoptype Associations dataset.
	ENCODE Histone Modification Site Profiles	histone modification site profiles with high histone modification abundance at ZMYND11 gene from the ENCODE Histone Modification Site Profiles dataset.
	ENCODE Transcription Factor Binding Site Profiles	transcription factor binding site profiles with transcription factor binding evidence at the promoter of ZMYND11 gene from the ENCODE Transcription Factor Binding Site Profiles dataset.
	ENCODE Transcription Factor Targets	transcription factors binding the promoter of ZMYND11 gene in ChIP-seq datasets from the ENCODE Transcription Factor Targets dataset.
	ESCAPE Omics Signatures of Genes and Proteins for Stem Cells	PubMedIDs of publications reporting gene signatures containing ZMYND11 from the ESCAPE Omics Signatures of Genes and Proteins for Stem Cells dataset.