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| Title / Description | Expression data from adult lungs isolated from TMEM100-deficient mice |
| Accession | GSE57256 |
| Summary | Tmem100 was identified as a downstream gene of BMP9-ALK1 signaling. The signaling has been implicated in hereditary hemorrhagic telangiectasia. This study was designed to identify affected genes in TMEM100-deficiect mice. Tmem100 was inactivated in adult Tmem1002loxP/2loxP;ROSA26+/CreER mice by tamoxifen tereatment. Microarray data was obtained from the lungs isolated from tamoxifen treated control and mutant mice. |
| Organism | Mus musculus |
| Platform | GPL6885 |
| Normalized | False |
| Parsed SOFT file |
| Differential expression method | Characteristic Direction |
| Cutoff | 500 |
| Cell | Lung |
| Perturbation | Tamoxifen |
| Disease | Hemorrhagic telangiectasia |
| Gene lists | Enrichr | L1000CDS2 | PAEA | |
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